Sofia Ahola
Koeln, NW, Germany
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 11 publications
Opa1 processing is dispensable in mouse development but is protective in mitochondrial cardiomyopathy.
Journal: Science advances
Published: August 02, 2024
Ferroptosis in mitochondrial cardiomyopathy.
Journal: Trends in cell biology
Published: April 13, 2023
The mitochondrial protease OMA1 acts as a metabolic safeguard upon nuclear DNA damage.
Journal: Cell reports
Published: September 23, 2022
OMA1-mediated integrated stress response protects against ferroptosis in mitochondrial cardiomyopathy.
Journal: Cell metabolism
Published: February 18, 2022
Mitochondrial Proteolysis and Metabolic Control.
Journal: Cold Spring Harbor perspectives in biology
Published: January 24, 2019
Loss of the mitochondrial i-AAA protease YME1L leads to ocular dysfunction and spinal axonopathy.
Journal: EMBO molecular medicine
Published: November 04, 2018
Metabolomes of mitochondrial diseases and inclusion body myositis patients: treatment targets and biomarkers.
Journal: EMBO molecular medicine
Published: October 31, 2018
Modified Atkins diet induces subacute selective ragged-red-fiber lysis in mitochondrial myopathy patients.
Journal: EMBO molecular medicine
Published: September 21, 2016
Mitochondrial DNA Replication Defects Disturb Cellular dNTP Pools and Remodel One-Carbon Metabolism.
Mitochondrial DNA Replication Defects Disturb Cellular dNTP Pools and Remodel One-Carbon Metabolism.
Journal: Cell metabolism
Published: September 10, 2015
Mitochondrial EFTs defects in juvenile-onset Leigh disease, ataxia, neuropathy, and optic atrophy.
Journal: Neurology
Published: July 20, 2014
MtDNA mutagenesis impairs elimination of mitochondria during erythroid maturation leading to enhanced erythrocyte destruction.
Journal: Nature communications
Published: July 09, 2014
Showing 1-11 of 11
Last Updated: 09/26/2026