Sonam Parakh

Sydney, NSW, Australia

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 26 publications

The Role of the Golgi Apparatus in Neurodegeneration.

The Role of the Golgi Apparatus in Neurodegeneration.

Journal: Sub-cellular biochemistry

Published: February 20, 2026

Cofilin hyperphosphorylation triggers TDP-43 pathology in sporadic amyotrophic lateral sclerosis.

Cofilin hyperphosphorylation triggers TDP-43 pathology in sporadic amyotrophic lateral sclerosis.

Journal: Brain : a journal of neurology

Published: July 31, 2025

Pathological forms of TDP-43 in amyotrophic lateral sclerosis (ALS) promote aberrant telomere elongation.

Pathological forms of TDP-43 in amyotrophic lateral sclerosis (ALS) promote aberrant telomere elongation.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: November 21, 2024

The Redox Activity of Protein Disulphide Isomerase Functions in Non-Homologous End-Joining Repair to Prevent DNA Damage.

The Redox Activity of Protein Disulphide Isomerase Functions in Non-Homologous End-Joining Repair to Prevent DNA Damage.

Journal: Aging cell

Published: September 23, 2024

Protein Disulfide Isomerase Endoplasmic Reticulum Protein 57 (ERp57) is Protective Against ALS-Associated Mutant TDP-43 in Neuronal Cells.

Protein Disulfide Isomerase Endoplasmic Reticulum Protein 57 (ERp57) is Protective Against ALS-Associated Mutant TDP-43 in Neuronal Cells.

Journal: Neuromolecular medicine

Published: November 16, 2023

ALS/FTD-associated mutation in cyclin F inhibits ER-Golgi trafficking, inducing ER stress, ERAD and Golgi fragmentation.

ALS/FTD-associated mutation in cyclin F inhibits ER-Golgi trafficking, inducing ER stress, ERAD and Golgi fragmentation.

Journal: Scientific reports

Published: April 03, 2023

Editorial: Restoring endoplasmic reticulum proteostasis to treat neurological disorders.

Editorial: Restoring endoplasmic reticulum proteostasis to treat neurological disorders.

Journal: Frontiers in pharmacology

Published: February 28, 2023

Redox dysregulation as a driver for DNA damage and its relationship to neurodegenerative diseases.

Redox dysregulation as a driver for DNA damage and its relationship to neurodegenerative diseases.

Journal: Translational neurodegeneration

Published: January 05, 2023

C9orf72-Associated Dipeptide Repeat Expansions Perturb ER-Golgi Vesicular Trafficking, Inducing Golgi Fragmentation and ER Stress, in ALS/FTD.

C9orf72-Associated Dipeptide Repeat Expansions Perturb ER-Golgi Vesicular Trafficking, Inducing Golgi Fragmentation and ER Stress, in ALS/FTD.

Journal: Molecular neurobiology

Published: December 22, 2022

The Mitochondrial-associated ER membrane (MAM) compartment and its dysregulation in Amyotrophic Lateral Sclerosis (ALS).

The Mitochondrial-associated ER membrane (MAM) compartment and its dysregulation in Amyotrophic Lateral Sclerosis (ALS).

Journal: Seminars in cell & developmental biology

Published: November 24, 2020

Emerging Evidence Highlighting the Importance of Redox Dysregulation in the Pathogenesis of Amyotrophic Lateral Sclerosis (ALS).

Emerging Evidence Highlighting the Importance of Redox Dysregulation in the Pathogenesis of Amyotrophic Lateral Sclerosis (ALS).

Journal: Frontiers in cellular neuroscience

Published: July 14, 2020

Correction to: the Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral Sclerosis.

Correction to: the Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral Sclerosis.

Journal: Journal of molecular neuroscience : MN

Published: June 13, 2020

Showing 1-12 of 26

Last Updated: 09/26/2026