Sonia Pagliardini
Milan, Italy
Last Updated: 09/26/2026
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Found 13 publications
COPZ1 depletion induces cell death through different mechanisms in in vitro murine models of thyroid cancer.
Journal: European thyroid journal
Published: December 19, 2025
Nrf2 shapes response to lysosomal inhibition of radiation-induced senescent thyroid cancer cells.
Journal: Endocrine-related cancer
Published: July 31, 2025
Genomic and transcriptomic analyses of thyroid cancers identify DICER1 somatic mutations in adult follicular-patterned RAS-like tumors.
Journal: Frontiers in endocrinology
Published: July 26, 2023
Correction: Oncogenic RAS-induced senescence in human primary thyrocytes: molecular effectors and inflammatory secretome involved.
Journal: Oncotarget
Published: October 16, 2022
Transcriptomic landscape of TIMP3 oncosuppressor activity in thyroid carcinoma.
Journal: Cancer cell international
Published: July 07, 2022
COPZ1 depletion in thyroid tumor cells triggers type I IFN response and immunogenic cell death.
Journal: Cancer letters
Published: October 10, 2019
Senescent thyrocytes and thyroid tumor cells induce M2-like macrophage polarization of human monocytes via a PGE2-dependent mechanism.
Journal: Journal of experimental & clinical cancer research : CR
Published: December 19, 2018
Mitosis perturbation by MASTL depletion impairs the viability of thyroid tumor cells.
Journal: Cancer letters
Published: August 28, 2018
Targeting COPZ1 non-oncogene addiction counteracts the viability of thyroid tumor cells.
Journal: Cancer letters
Published: July 11, 2017
Identification of thyroid tumor cell vulnerabilities through a siRNA-based functional screening.
Journal: Oncotarget
Published: June 06, 2015
Last Updated: 09/26/2026