Stefan Gaubatz
Wuerzburg, BY 97074, Germany
Last Updated: 09/26/2026
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Found 18 publications
Oncogenic YAP sensitizes cells to CHK1 inhibition via CDK4/6 driven G1 acceleration.
Journal: EMBO reports
Published: February 11, 2025
Expression of the cytokinesis regulator PRC1 results in p53-pathway activation in A549 cells but does not directly regulate gene expression in the nucleus.
Journal: Cell cycle (Georgetown, Tex.)
Published: September 22, 2022
Oncogenic YAP mediates changes in chromatin accessibility and activity that drive cell cycle gene expression and cell migration.
Journal: Nucleic acids research
Published: September 06, 2022
IQGAP3, a YAP Target, Is Required for Proper Cell-Cycle Progression and Genome Stability.
Journal: Molecular cancer research : MCR
Published: July 20, 2020
Interaction of YAP with the Myb-MuvB (MMB) complex defines a transcriptional program to promote the proliferation of cardiomyocytes.
Journal: PLoS genetics
Published: January 08, 2020
One way to rule them all: G2/M gene regulation by oncogenic YAP1 and B-MYB.
Journal: Molecular & cellular oncology
Published: July 15, 2019
The Myb-MuvB Complex Is Required for YAP-Dependent Transcription of Mitotic Genes.
Journal: Cell reports
Published: December 18, 2018
GAS2L3: Coordinator of cardiomyocyte cytokinesis?
Journal: Cell cycle (Georgetown, Tex.)
Published: September 23, 2017
The microtubule-associated protein PRC1 is a potential therapeutic target for lung cancer.
Journal: Oncotarget
Published: August 05, 2017
Deletion of Gas2l3 in mice leads to specific defects in cardiomyocyte cytokinesis during development.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: July 13, 2017
Central spindle proteins and mitotic kinesins are direct transcriptional targets of MuvB, B-MYB and FOXM1 in breast cancer cell lines and are potential targets for therapy.
Journal: Oncotarget
Published: October 04, 2016
Last Updated: 09/26/2026