Stefan Rebhandl
Salzburg, 5020, Austria
Last Updated: 09/26/2026
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Found 12 publications
SAMHD1 restrains aberrant nucleotide insertions at repair junctions generated by DNA end joining.
Journal: Nucleic acids research
Published: August 13, 2020
Imprecision and DNA Break Repair Biased towards Incompatible End Joining in Leukemia.
Journal: Molecular cancer research : MCR
Published: July 13, 2017
CD1d expression on chronic lymphocytic leukemia B cells affects disease progression and induces T cell skewing in CD8 positive and CD4CD8 double negative T cells.
Journal: Oncotarget
Published: August 25, 2015
AIDing cancer treatment: Reducing AID activity via HSP90 inhibition.
Journal: European journal of immunology
Published: June 08, 2015
Chronic lymphocytic leukaemia induces an exhausted T cell phenotype in the TCL1 transgenic mouse model.
Journal: British journal of haematology
Published: January 28, 2015
APOBEC3 signature mutations in chronic lymphocytic leukemia.
Journal: Leukemia
Published: May 21, 2014
Chemotherapy-induced augmentation of T cells expressing inhibitory receptors is reversed by treatment with lenalidomide in chronic lymphocytic leukemia.
Journal: Haematologica
Published: February 25, 2014
Targeting proliferation of chronic lymphocytic leukemia (CLL) cells through KCa3.1 blockade.
Journal: Leukemia
Published: January 21, 2014
AID induces intraclonal diversity and genomic damage in CD86(+) chronic lymphocytic leukemia cells.
Journal: European journal of immunology
Published: December 20, 2013
Alternative splice variants of AID are not stoichiometrically present at the protein level in chronic lymphocytic leukemia.
Journal: European journal of immunology
Published: July 01, 2013
Showing 1-12 of 12
Last Updated: 09/26/2026