Steffen Hermans
Koeln, NW, Germany
Last Updated: 09/26/2026
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Found 8 publications
Opa1 processing is dispensable in mouse development but is protective in mitochondrial cardiomyopathy.
Journal: Science advances
Published: August 02, 2024
OMA1-mediated integrated stress response protects against ferroptosis in mitochondrial cardiomyopathy.
Journal: Cell metabolism
Published: February 18, 2022
Cellular pyrimidine imbalance triggers mitochondrial DNA-dependent innate immunity.
Journal: Nature metabolism
Published: June 08, 2020
CLPP deficiency protects against metabolic syndrome but hinders adaptive thermogenesis.
Journal: EMBO reports
Published: September 05, 2017
DARS2 protects against neuroinflammation and apoptotic neuronal loss, but is dispensable for myelin producing cells.
Journal: Human molecular genetics
Published: April 28, 2017
Loss of CLPP alleviates mitochondrial cardiomyopathy without affecting the mammalian UPRmt.
Journal: EMBO reports
Published: January 21, 2016
Showing 1-8 of 8
Last Updated: 09/26/2026