Stella D. Nyamoya

Aachen, NW, Germany

Last Updated: 09/26/2026

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Found 18 publications

Lipocalin 2 attenuates oligodendrocyte loss and immune cell infiltration in mouse models for multiple sclerosis.

Lipocalin 2 attenuates oligodendrocyte loss and immune cell infiltration in mouse models for multiple sclerosis.

Journal: Glia
Published: January 11, 2022

Laquinimod Supports Remyelination in Non-Supportive Environments.

Laquinimod Supports Remyelination in Non-Supportive Environments.

Journal: Cells
Published: October 10, 2019

Nrf2 deficiency increases oligodendrocyte loss, demyelination, neuroinflammation and axonal damage in an MS animal model.

Nrf2 deficiency increases oligodendrocyte loss, demyelination, neuroinflammation and axonal damage in an MS animal model.

Journal: Metabolic brain disease
Published: March 21, 2019

Expression of Translocator Protein and [18F]-GE180 Ligand Uptake in Multiple Sclerosis Animal Models.

Expression of Translocator Protein and [18F]-GE180 Ligand Uptake in Multiple Sclerosis Animal Models.

Journal: Cells
Published: November 20, 2018

Oligodendrocyte degeneration and concomitant microglia activation directs peripheral immune cells into the forebrain.

Oligodendrocyte degeneration and concomitant microglia activation directs peripheral immune cells into the forebrain.

Journal: Neurochemistry international
Published: July 31, 2018

Mitochondrial Impairment in Oligodendroglial Cells Induces Cytokine Expression and Signaling.

Mitochondrial Impairment in Oligodendroglial Cells Induces Cytokine Expression and Signaling.

Journal: Journal of molecular neuroscience : MN
Published: July 25, 2018

Cuprizone-induced graded oligodendrocyte vulnerability is regulated by the transcription factor DNA damage-inducible transcript 3.

Cuprizone-induced graded oligodendrocyte vulnerability is regulated by the transcription factor DNA damage-inducible transcript 3.

Journal: Glia
Published: April 03, 2018

Nrf2 Signaling in Sodium Azide-Treated Oligodendrocytes Restores Mitochondrial Functions.

Nrf2 Signaling in Sodium Azide-Treated Oligodendrocytes Restores Mitochondrial Functions.

Journal: Journal of molecular neuroscience : MN
Published: March 19, 2018

G-Protein-Coupled Receptor Gpr17 Expression in Two Multiple Sclerosis Remyelination Models.

G-Protein-Coupled Receptor Gpr17 Expression in Two Multiple Sclerosis Remyelination Models.

Journal: Molecular neurobiology
Published: January 22, 2018

Phosphatidylcholine 36:1 concentration decreases along with demyelination in the cuprizone animal model and in post-mortem multiple sclerosis brain tissue.

Phosphatidylcholine 36:1 concentration decreases along with demyelination in the cuprizone animal model and in post-mortem multiple sclerosis brain tissue.

Journal: Journal of neurochemistry
Published: December 17, 2017

Toll-Like Receptor 2-Mediated Glial Cell Activation in a Mouse Model of Cuprizone-Induced Demyelination.

Toll-Like Receptor 2-Mediated Glial Cell Activation in a Mouse Model of Cuprizone-Induced Demyelination.

Journal: Molecular neurobiology
Published: July 18, 2017

Combination of cuprizone and experimental autoimmune encephalomyelitis to study inflammatory brain lesion formation and progression.

Combination of cuprizone and experimental autoimmune encephalomyelitis to study inflammatory brain lesion formation and progression.

Journal: Glia
Published: April 03, 2017
Showing 1-12 of 18

Last Updated: 09/26/2026