Stephanie Erschfeld
Aachen, NW, Germany
Last Updated: 09/26/2026
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Found 11 publications
Selective genetic inactivation of Caspase 8 in hepatocytes ameliorates progression of MASH following Jnk deficiency.
Journal: Hepatology (Baltimore, Md.)
Published: November 22, 2024
Lack of Cyclin E1 in hepatocytes aggravates ethanol-induced liver injury and hepatic steatosis in experimental murine model of acute and chronic alcohol-associated liver disease.
Journal: Biochimica et biophysica acta. Molecular basis of disease
Published: September 13, 2022
ACSL4-dependent ferroptosis does not represent a tumor-suppressive mechanism but ACSL4 rather promotes liver cancer progression.
Journal: Cell death & disease
Published: May 27, 2022
The Inhibitory T Cell Receptors PD1 and 2B4 Are Differentially Regulated on CD4 and CD8 T Cells in a Mouse Model of Non-alcoholic Steatohepatitis.
Journal: Frontiers in pharmacology
Published: December 26, 2018
Genetic Nrf2 Overactivation Inhibits the Deleterious Effects Induced by Hepatocyte-Specific c-met Deletion during the Progression of NASH.
Journal: Oxidative medicine and cellular longevity
Published: December 12, 2016
Hepatocyte-specific Keap1 deletion reduces liver steatosis but not inflammation during non-alcoholic steatohepatitis development.
Journal: Free radical biology & medicine
Published: September 29, 2015
The Anti-Microbial Peptide LL-37/CRAMP Is Elevated in Patients with Liver Diseases and Acts as a Protective Factor during Mouse Liver Injury.
Journal: Digestion
Published: March 27, 2014
Hepatic tissue environment in NEMO-deficient mice critically regulates positive selection of donor cells after hepatocyte transplantation.
Journal: PloS one
Published: January 29, 2014
Lack of hepatic c-Met and gp130 expression is associated with an impaired antibacterial response and higher lethality after bile duct ligation.
Journal: Laboratory investigation; a journal of technical methods and pathology
Published: September 19, 2012
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Last Updated: 09/26/2026