Steve Gerondakis
Wellington Road
Clayton, VIC 3800, Australia
Clayton, VIC 3800, Australia
Last Updated: 09/26/2026
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Found 25 publications
Mice lacking NF-ĸB1 undergo premature ovarian aging.
Journal: Reproductive biology and endocrinology : RB&E
Published: March 10, 2026
c-Rel Is Required for IL-33-Dependent Activation of ILC2s.
Journal: Frontiers in immunology
Published: February 15, 2021
c-Rel employs multiple mechanisms to promote the thymic development and peripheral function of regulatory T cells in mice.
Journal: European journal of immunology
Published: July 23, 2020
Rel-Dependent Immune and Central Nervous System Mechanisms Control Viral Replication and Inflammation during Mouse Herpes Simplex Encephalitis.
Journal: Journal of immunology (Baltimore, Md. : 1950)
Published: January 16, 2018
Corrigendum: NF-κB control of T cell development.
Journal: Nature immunology
Published: September 20, 2017
Loss of NF-κB1 Causes Gastric Cancer with Aberrant Inflammation and Expression of Immune Checkpoint Regulators in a STAT-1-Dependent Manner.
Journal: Immunity
Published: August 08, 2017
Evidence that TLR4 Is Not a Receptor for Saturated Fatty Acids but Mediates Lipid-Induced Inflammation by Reprogramming Macrophage Metabolism.
Journal: Cell metabolism
Published: June 23, 2017
The Bacterial Peptidoglycan-Sensing Molecules NOD1 and NOD2 Promote CD8+ Thymocyte Selection.
Journal: Journal of immunology (Baltimore, Md. : 1950)
Published: October 13, 2016
The NF-κB transcription factor RelA is required for the tolerogenic function of Foxp3(+) regulatory T cells.
Journal: Journal of autoimmunity
Published: December 17, 2015
Understanding the Roles of the NF-κB Pathway in Regulatory T Cell Development, Differentiation and Function.
Journal: Progress in molecular biology and translational science
Published: November 29, 2015
NFκB1 is essential to prevent the development of multiorgan autoimmunity by limiting IL-6 production in follicular B cells.
Journal: The Journal of experimental medicine
Published: July 20, 2015
Impact of loss of NF-κB1, NF-κB2 or c-REL on SLE-like autoimmune disease and lymphadenopathy in Fas(lpr/lpr) mutant mice.
Journal: Immunology and cell biology
Published: April 30, 2015
Last Updated: 09/26/2026