Steven B. Marston

London, ENG, United Kingdom

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 66 publications

5,7-Dimethoxychroman-3-yl 4-methoxybenzoate Has a Unique Effect upon the Modulation of Mutant Cardiac Muscle Thin Filament Function and Dynamics due to Phosphorylation of Troponin I.

5,7-Dimethoxychroman-3-yl 4-methoxybenzoate Has a Unique Effect upon the Modulation of Mutant Cardiac Muscle Thin Filament Function and Dynamics due to Phosphorylation of Troponin I.

Journal: ACS pharmacology & translational science

Published: February 27, 2025

Nutraceuticals silybin B, resveratrol, and epigallocatechin-3 gallate-bind to cardiac muscle troponin to restore the loss of lusitropy caused by cardiomyopathy mutations in vitro, in vivo, and in silico.

Nutraceuticals silybin B, resveratrol, and epigallocatechin-3 gallate-bind to cardiac muscle troponin to restore the loss of lusitropy caused by cardiomyopathy mutations in vitro, in vivo, and in silico.

Journal: Frontiers in physiology

Published: September 01, 2024

Modulation of Structure and Dynamics of Cardiac Troponin by Phosphorylation and Mutations Revealed by Molecular Dynamics Simulations.

Modulation of Structure and Dynamics of Cardiac Troponin by Phosphorylation and Mutations Revealed by Molecular Dynamics Simulations.

Journal: The journal of physical chemistry. B

Published: October 04, 2023

Suppression of lusitropy as a disease mechanism in cardiomyopathies.

Suppression of lusitropy as a disease mechanism in cardiomyopathies.

Journal: Frontiers in cardiovascular medicine

Published: October 26, 2022

Recent studies of the molecular mechanism of lusitropy due to phosphorylation of cardiac troponin I by protein kinase A.

Recent studies of the molecular mechanism of lusitropy due to phosphorylation of cardiac troponin I by protein kinase A.

Journal: Journal of muscle research and cell motility

Published: June 19, 2022

Modulation of cardiac thin filament structure by phosphorylated troponin-I analyzed by protein-protein docking and molecular dynamics simulation.

Modulation of cardiac thin filament structure by phosphorylated troponin-I analyzed by protein-protein docking and molecular dynamics simulation.

Journal: Archives of biochemistry and biophysics

Published: January 31, 2022

Force Measurements From Myofibril to Filament.

Force Measurements From Myofibril to Filament.

Journal: Frontiers in physiology

Published: November 17, 2021

Small Molecules acting on Myofilaments as Treatments for Heart and Skeletal Muscle Diseases.

Small Molecules acting on Myofilaments as Treatments for Heart and Skeletal Muscle Diseases.

Journal: International journal of molecular sciences

Published: November 23, 2020

Distinct hypertrophic cardiomyopathy genotypes result in convergent sarcomeric proteoform profiles revealed by top-down proteomics.

Distinct hypertrophic cardiomyopathy genotypes result in convergent sarcomeric proteoform profiles revealed by top-down proteomics.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: September 24, 2020

Donor hearts in the Sydney Heart Bank: reliable control but is it 'normal' heart?

Donor hearts in the Sydney Heart Bank: reliable control but is it 'normal' heart?

Journal: Biophysical reviews

Published: May 01, 2020

Approaches to High-Throughput Analysis of Cardiomyocyte Contractility.

Approaches to High-Throughput Analysis of Cardiomyocyte Contractility.

Journal: Frontiers in physiology

Published: January 20, 2020

Pressure Overload Is Associated With Low Levels of Troponin I and Myosin Binding Protein C Phosphorylation in the Hearts of Patients With Aortic Stenosis.

Pressure Overload Is Associated With Low Levels of Troponin I and Myosin Binding Protein C Phosphorylation in the Hearts of Patients With Aortic Stenosis.

Journal: Frontiers in physiology

Published: December 19, 2019

Showing 1-12 of 66

Last Updated: 09/26/2026