Steven J. Schwulst

Steven J. Schwulst

General Surgery

259 E Erie St Ste 1600
Lavin Family Pavilion
Chicago, IL 60611

Last Updated: 09/26/2026

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Found 35 publications

Sexual Dimorphism in the Microglial Inflammatory Response After Traumatic Brain Injury.

Sexual Dimorphism in the Microglial Inflammatory Response After Traumatic Brain Injury.

Journal: Shock (Augusta, Ga.)

Published: August 28, 2026

Management of Acute Cholecystitis in a Patient With Eisenmenger Syndrome and Abdominal Heterotaxy: A Case Report.

Management of Acute Cholecystitis in a Patient With Eisenmenger Syndrome and Abdominal Heterotaxy: A Case Report.

Journal: The American journal of case reports

Published: February 06, 2026

Depletion of Peripheral Monocytes Alters Long-Term Gene Expression in Microglia in a Murine Model of Traumatic Brain Injury.

Depletion of Peripheral Monocytes Alters Long-Term Gene Expression in Microglia in a Murine Model of Traumatic Brain Injury.

Journal: Shock (Augusta, Ga.)

Published: September 17, 2025

Short-Chain Fatty Acid Supplementation After Traumatic Brain Injury Attenuates Neurologic Injury Via the Gut-Brain-Microglia Axis.

Short-Chain Fatty Acid Supplementation After Traumatic Brain Injury Attenuates Neurologic Injury Via the Gut-Brain-Microglia Axis.

Journal: Shock (Augusta, Ga.)

Published: September 17, 2025

antiCD49d Ab treatment ameliorates age-associated inflammatory response and mitigates CD8+ T-cell cytotoxicity after traumatic brain injury.

antiCD49d Ab treatment ameliorates age-associated inflammatory response and mitigates CD8+ T-cell cytotoxicity after traumatic brain injury.

Journal: bioRxiv : the preprint server for biology

Published: July 01, 2024

Factors associated with unfavorable outcomes in older patients with traumatic brain injury: analysis from the "All of Us" research program.

Factors associated with unfavorable outcomes in older patients with traumatic brain injury: analysis from the "All of Us" research program.

Journal: Frontiers in neurology

Published: June 24, 2024

Anti-CD49d Ab treatment ameliorates age-associated inflammatory response and mitigates CD8+ T-cell cytotoxicity after traumatic brain injury.

Anti-CD49d Ab treatment ameliorates age-associated inflammatory response and mitigates CD8+ T-cell cytotoxicity after traumatic brain injury.

Journal: Journal of neuroinflammation

Published: June 20, 2024

The gut microbiome controls reactive astrocytosis during Aβ amyloidosis via propionate-mediated regulation of IL-17.

The gut microbiome controls reactive astrocytosis during Aβ amyloidosis via propionate-mediated regulation of IL-17.

Journal: The Journal of clinical investigation

Published: March 04, 2024

ANTI-CD49D ANTIBODY TREATMENT IMPROVES SURVIVAL AND ATTENUATES NEUROCOGNITIVE DEFICITS AFTER TRAUMATIC BRAIN INJURY IN AGED MICE.

ANTI-CD49D ANTIBODY TREATMENT IMPROVES SURVIVAL AND ATTENUATES NEUROCOGNITIVE DEFICITS AFTER TRAUMATIC BRAIN INJURY IN AGED MICE.

Journal: Shock (Augusta, Ga.)

Published: November 27, 2023

MICROGLIA AND INFILTRATING T-CELLS ADOPT LONG-TERM, AGE-SPECIFIC, TRANSCRIPTIONAL CHANGES AFTER TRAUMATIC BRAIN INJURY IN MICE.

MICROGLIA AND INFILTRATING T-CELLS ADOPT LONG-TERM, AGE-SPECIFIC, TRANSCRIPTIONAL CHANGES AFTER TRAUMATIC BRAIN INJURY IN MICE.

Journal: Shock (Augusta, Ga.)

Published: February 02, 2023

POSTINJURY FECAL MICROBIOME TRANSPLANT DECREASES LESION SIZE AND NEUROINFLAMMATION IN TRAUMATIC BRAIN INJURY.

POSTINJURY FECAL MICROBIOME TRANSPLANT DECREASES LESION SIZE AND NEUROINFLAMMATION IN TRAUMATIC BRAIN INJURY.

Journal: Shock (Augusta, Ga.)

Published: October 18, 2022

Fecal Microbiota Transfer Attenuates Gut Dysbiosis and Functional Deficits After Traumatic Brain Injury.

Fecal Microbiota Transfer Attenuates Gut Dysbiosis and Functional Deficits After Traumatic Brain Injury.

Journal: Shock (Augusta, Ga.)

Published: June 27, 2022

Showing 1-12 of 35

Last Updated: 09/26/2026