Sukhpal Prehar
Manchester, ENG M139PL, United Kingdom
Last Updated: 09/26/2026
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Found 28 publications
Loss of Miro1 drives cardiac and mitochondrial dysfunction exacerbated by metabolic stress.
Journal: Journal of molecular and cellular cardiology
Published: January 09, 2026
Treatment with αvβ3-integrin-specific 29P attenuates pressure-overload induced cardiac remodelling after transverse aortic constriction in mice.
Journal: Journal of molecular and cellular cardiology plus
Published: November 17, 2023
Expression of foetal gene Pontin is essential in protecting heart against pathological remodelling and cardiomyopathy.
Journal: Nature communications
Published: September 12, 2023
Micro RNA-411 Expression Improves Cardiac Phenotype Following Myocardial Infarction in Mice.
Journal: JACC. Basic to translational science
Published: September 07, 2021
Cardiomyocyte-specific loss of plasma membrane calcium ATPase 1 impacts cardiac rhythm and is associated with ventricular repolarisation dysfunction.
Journal: Journal of molecular and cellular cardiology
Published: August 17, 2021
Differential remodelling of mitochondrial subpopulations and mitochondrial dysfunction are a feature of early stage diabetes.
Journal: Scientific reports
Published: July 08, 2021
PMCA4 inhibition does not affect cardiac remodelling following myocardial infarction, but may reduce susceptibility to arrhythmia.
Journal: Scientific reports
Published: May 21, 2020
Signaling via the Interleukin-10 Receptor Attenuates Cardiac Hypertrophy in Mice During Pressure Overload, but not Isoproterenol Infusion.
Journal: Frontiers in pharmacology
Published: May 05, 2020
Cardiac hypertrophy or failure? - A systematic evaluation of the transverse aortic constriction model in C57BL/6NTac and C57BL/6J substrains.
Journal: Current research in physiology
Published: July 31, 2019
Cardiac mitochondrial function depends on BUD23 mediated ribosome programming.
Journal: eLife
Published: July 30, 2019
Last Updated: 09/26/2026