Sushil C. Regmi

Gyeongsan-si, Republic of Korea

Last Updated: 09/26/2026

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Found 14 publications

TAK1 is essential for endothelial barrier maintenance and repair after lung vascular injury.

TAK1 is essential for endothelial barrier maintenance and repair after lung vascular injury.

Journal: Molecular biology of the cell

Published: March 24, 2022

Synthesis and activity of N-(5-hydroxy-3,4,6-trimethylpyridin-2-yl)acetamide analogues as anticolitis agents via dual inhibition of TNF-α- and IL-6-induced cell adhesions.

Synthesis and activity of N-(5-hydroxy-3,4,6-trimethylpyridin-2-yl)acetamide analogues as anticolitis agents via dual inhibition of TNF-α- and IL-6-induced cell adhesions.

Journal: Bioorganic & medicinal chemistry letters

Published: February 04, 2021

EphB1 interaction with caveolin-1 in endothelial cells modulates caveolae biogenesis.

EphB1 interaction with caveolin-1 in endothelial cells modulates caveolae biogenesis.

Journal: Molecular biology of the cell

Published: April 03, 2020

Deubiquitinase function of A20 maintains and repairs endothelial barrier after lung vascular injury.

Deubiquitinase function of A20 maintains and repairs endothelial barrier after lung vascular injury.

Journal: Cell death discovery

Published: March 12, 2018

Pyk2 phosphorylation of VE-PTP downstream of STIM1-induced Ca2+ entry regulates disassembly of adherens junctions.

Pyk2 phosphorylation of VE-PTP downstream of STIM1-induced Ca2+ entry regulates disassembly of adherens junctions.

Journal: American journal of physiology. Lung cellular and molecular physiology

Published: January 06, 2017

Dual Inhibition of NOX2 and Receptor Tyrosine Kinase by BJ-1301 Enhances Anticancer Therapy Efficacy via Suppression of Autocrine-Stimulatory Factors in Lung Cancer.

Dual Inhibition of NOX2 and Receptor Tyrosine Kinase by BJ-1301 Enhances Anticancer Therapy Efficacy via Suppression of Autocrine-Stimulatory Factors in Lung Cancer.

Journal: Molecular cancer therapeutics

Published: December 28, 2016

Serotonin disturbs colon epithelial tolerance of commensal E. coli by increasing NOX2-derived superoxide.

Serotonin disturbs colon epithelial tolerance of commensal E. coli by increasing NOX2-derived superoxide.

Journal: Free radical biology & medicine

Published: October 17, 2016

Tryptophan hydroxylase 1 and 5-HT7 receptor preferentially expressed in triple-negative breast cancer promote cancer progression through autocrine serotonin signaling.

Tryptophan hydroxylase 1 and 5-HT7 receptor preferentially expressed in triple-negative breast cancer promote cancer progression through autocrine serotonin signaling.

Journal: Molecular cancer

Published: June 23, 2016

BJ-1108, a 6-Amino-2,4,5-Trimethylpyridin-3-ol Analog, Inhibits Serotonin-Induced Angiogenesis and Tumor Growth through PI3K/NOX Pathway.

BJ-1108, a 6-Amino-2,4,5-Trimethylpyridin-3-ol Analog, Inhibits Serotonin-Induced Angiogenesis and Tumor Growth through PI3K/NOX Pathway.

Journal: PloS one

Published: October 17, 2015

The Anti-Tumor Activity of Succinyl Macrolactin A Is Mediated through the β-Catenin Destruction Complex via the Suppression of Tankyrase and PI3K/Akt.

The Anti-Tumor Activity of Succinyl Macrolactin A Is Mediated through the β-Catenin Destruction Complex via the Suppression of Tankyrase and PI3K/Akt.

Journal: PloS one

Published: June 11, 2015

NOX1 to NOX2 switch deactivates AMPK and induces invasive phenotype in colon cancer cells through overexpression of MMP-7.

NOX1 to NOX2 switch deactivates AMPK and induces invasive phenotype in colon cancer cells through overexpression of MMP-7.

Journal: Molecular cancer

Published: December 04, 2014

Anti-angiogenic activity of macrolactin A and its succinyl derivative is mediated through inhibition of class I PI3K activity and its signaling.

Anti-angiogenic activity of macrolactin A and its succinyl derivative is mediated through inhibition of class I PI3K activity and its signaling.

Journal: Archives of pharmacal research

Published: November 10, 2014

Showing 1-12 of 14

Last Updated: 09/26/2026