Sylvie Dussault

Montreal, QC H2X0A9, Canada

Last Updated: 09/26/2026

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Found 17 publications

Hypercholesterolemia Impairs the Expression of Angiogenic MicroRNAs in Extracellular Vesicles Within Ischemic Skeletal Muscles.

Hypercholesterolemia Impairs the Expression of Angiogenic MicroRNAs in Extracellular Vesicles Within Ischemic Skeletal Muscles.

Journal: Non-coding RNA

Published: November 07, 2025

Selective Enrichment of Angiomirs in Extracellular Vesicles Released from Ischemic Skeletal Muscles: Potential Role in Angiogenesis and Neovascularization.

Selective Enrichment of Angiomirs in Extracellular Vesicles Released from Ischemic Skeletal Muscles: Potential Role in Angiogenesis and Neovascularization.

Journal: Cells

Published: May 24, 2024

Endothelial extracellular vesicle miR-423-5p regulates microvascular homeostasis and renal function after ischemia-reperfusion injury.

Endothelial extracellular vesicle miR-423-5p regulates microvascular homeostasis and renal function after ischemia-reperfusion injury.

Journal: JCI insight

Published: April 11, 2024

Stimulation of soluble guanylate cyclase activity with riociguat promotes angiogenesis and improves neovascularization after limb ischemia.

Stimulation of soluble guanylate cyclase activity with riociguat promotes angiogenesis and improves neovascularization after limb ischemia.

Journal: Atherosclerosis

Published: June 03, 2022

14-3-3ζ mediates an alternative, non-thermogenic mechanism in male mice to reduce heat loss and improve cold tolerance.

14-3-3ζ mediates an alternative, non-thermogenic mechanism in male mice to reduce heat loss and improve cold tolerance.

Journal: Molecular metabolism

Published: June 15, 2020

MicroRNA Expression Profiling of Bone Marrow-Derived Proangiogenic Cells (PACs) in a Mouse Model of Hindlimb Ischemia: Modulation by Classical Cardiovascular Risk Factors.

MicroRNA Expression Profiling of Bone Marrow-Derived Proangiogenic Cells (PACs) in a Mouse Model of Hindlimb Ischemia: Modulation by Classical Cardiovascular Risk Factors.

Journal: Frontiers in genetics

Published: April 14, 2020

Reduced expression of microRNA-130a promotes endothelial cell senescence and age-dependent impairment of neovascularization.

Reduced expression of microRNA-130a promotes endothelial cell senescence and age-dependent impairment of neovascularization.

Journal: Aging

Published: November 24, 2019

Forced expression of microRNA-146b reduces TRAF6-dependent inflammation and improves ischemia-induced neovascularization in hypercholesterolemic conditions.

Forced expression of microRNA-146b reduces TRAF6-dependent inflammation and improves ischemia-induced neovascularization in hypercholesterolemic conditions.

Journal: Atherosclerosis

Published: January 24, 2019

Neonatal exposure to high oxygen levels leads to impaired ischemia-induced neovascularization in adulthood.

Neonatal exposure to high oxygen levels leads to impaired ischemia-induced neovascularization in adulthood.

Journal: Scientific reports

Published: April 13, 2017

Reduced expression of let-7f activates TGF-β/ALK5 pathway and leads to impaired ischaemia-induced neovascularization after cigarette smoke exposure.

Reduced expression of let-7f activates TGF-β/ALK5 pathway and leads to impaired ischaemia-induced neovascularization after cigarette smoke exposure.

Journal: Journal of cellular and molecular medicine

Published: September 25, 2016

MicroRNA-150 Modulates Ischemia-Induced Neovascularization in Atherosclerotic Conditions.

MicroRNA-150 Modulates Ischemia-Induced Neovascularization in Atherosclerotic Conditions.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: August 10, 2016

Tyrosine Phosphatase PTPRJ/DEP-1 Is an Essential Promoter of Vascular Permeability, Angiogenesis, and Tumor Progression.

Tyrosine Phosphatase PTPRJ/DEP-1 Is an Essential Promoter of Vascular Permeability, Angiogenesis, and Tumor Progression.

Journal: Cancer research

Published: April 13, 2016

Showing 1-12 of 17

Last Updated: 09/26/2026