Takahiro Eguchi

Tokyo, 1088639, Japan

Last Updated: 09/26/2026

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Found 11 publications

Calcium-binding protein 7 expressed in muscle negatively regulates age-related degeneration of neuromuscular junctions in mice.

Calcium-binding protein 7 expressed in muscle negatively regulates age-related degeneration of neuromuscular junctions in mice.

Journal: iScience

Published: August 30, 2023

Adeno-associated virus-mediated expression of an inactive CaMKIIβ mutant enhances muscle mass and strength in mice.

Adeno-associated virus-mediated expression of an inactive CaMKIIβ mutant enhances muscle mass and strength in mice.

Journal: Biochemical and biophysical research communications

Published: October 23, 2021

DOK7 Gene Therapy Enhances Neuromuscular Junction Innervation and Motor Function in Aged Mice.

DOK7 Gene Therapy Enhances Neuromuscular Junction Innervation and Motor Function in Aged Mice.

Journal: iScience

Published: April 01, 2020

Overexpression of Dok-7 in skeletal muscle enhances neuromuscular transmission with structural alterations of neuromuscular junctions: Implications in robustness of neuromuscular transmission.

Overexpression of Dok-7 in skeletal muscle enhances neuromuscular transmission with structural alterations of neuromuscular junctions: Implications in robustness of neuromuscular transmission.

Journal: Biochemical and biophysical research communications

Published: November 19, 2019

Roles of Collagen XXV and Its Putative Receptors PTPσ/δ in Intramuscular Motor Innervation and Congenital Cranial Dysinnervation Disorder.

Roles of Collagen XXV and Its Putative Receptors PTPσ/δ in Intramuscular Motor Innervation and Congenital Cranial Dysinnervation Disorder.

Journal: Cell reports

Published: March 15, 2019

Molecular mechanisms underlying the formation and maintenance of neuromuscular junctions and a possible therapeutic approach.

Molecular mechanisms underlying the formation and maintenance of neuromuscular junctions and a possible therapeutic approach.

Journal: Clinical calcium

Published: February 25, 2017

Postnatal knockdown of dok-7 gene expression in mice causes structural defects in neuromuscular synapses and myasthenic pathology.

Postnatal knockdown of dok-7 gene expression in mice causes structural defects in neuromuscular synapses and myasthenic pathology.

Journal: Genes to cells : devoted to molecular & cellular mechanisms

Published: February 19, 2016

Data supporting mitochondrial morphological changes by SPG13-associated HSPD1 mutants.

Data supporting mitochondrial morphological changes by SPG13-associated HSPD1 mutants.

Journal: Data in brief

Published: October 10, 2015

Hypomyelinating leukodystrophy-associated missense mutation in HSPD1 blunts mitochondrial dynamics.

Hypomyelinating leukodystrophy-associated missense mutation in HSPD1 blunts mitochondrial dynamics.

Journal: Biochemical and biophysical research communications

Published: April 20, 2015

Hypomyelinating leukodystrophy-associated missense mutant of FAM126A/hyccin/DRCTNNB1A aggregates in the endoplasmic reticulum.

Hypomyelinating leukodystrophy-associated missense mutant of FAM126A/hyccin/DRCTNNB1A aggregates in the endoplasmic reticulum.

Journal: Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia

Published: February 12, 2013

Paxillin is the target of c-Jun N-terminal kinase in Schwann cells and regulates migration.

Paxillin is the target of c-Jun N-terminal kinase in Schwann cells and regulates migration.

Journal: Cellular signalling

Published: June 01, 2012

Showing 1-11 of 11

Last Updated: 09/26/2026