Takanori Yamada
Fuchu, 1838509, Japan
Last Updated: 09/26/2026
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Found 12 publications
Persistent γ-H2AX Formation and Expression of Stem Cell Markers in N-Butyl-N-(4-Hydroxybutyl)Nitrosamine-Induced Bladder Carcinogenesis in Rats.
Journal: Toxicological sciences : an official journal of the Society of Toxicology
Published: June 30, 2022
o-Anisidine Dimer, 2-Methoxy-N4-(2-methoxyphenyl) Benzene-1,4-diamine, in Rat Urine Associated with Urinary bladder Carcinogenesis.
Journal: Chemical research in toxicology
Published: February 15, 2021
Correction to A Novel o-Toluidine Metabolite in Rat Urine Associated with Urinary Bladder Carcinogenesis.
Journal: Chemical research in toxicology
Published: November 17, 2020
Expression of stem cell markers as useful complementary factors in the early detection of urinary bladder carcinogens by immunohistochemistry for γ-H2AX.
Journal: Archives of toxicology
Published: September 21, 2020
Neuromuscular and vascular hamartoma of the small intestine in an F344 rat.
Journal: Journal of toxicologic pathology
Published: August 28, 2020
Specific expression of survivin, SOX9, and CD44 in renal tubules in adaptive and maladaptive repair processes after acute kidney injury in rats.
Journal: Journal of applied toxicology : JAT
Published: June 17, 2020
Novel o-Toluidine Metabolite in Rat Urine Associated with Urinary Bladder Carcinogenesis.
Journal: Chemical research in toxicology
Published: April 29, 2020
Comprehensive expression analysis of mRNA and microRNA for the investigation of compensatory mechanisms in the rat kidney after unilateral nephrectomy.
Journal: Journal of applied toxicology : JAT
Published: February 05, 2020
Dose dependency of γ-H2AX formation in the rat urinary bladder treated with genotoxic and nongenotoxic bladder carcinogens.
Journal: Journal of applied toxicology : JAT
Published: February 02, 2020
Distinct differences in the mechanisms of mucosal damage and γ-H2AX formation in the rat urinary bladder treated with o-toluidine and o-anisidine.
Journal: Archives of toxicology
Published: September 20, 2018
Diphenylarsinic acid, a chemical warfare-related neurotoxicant, promotes liver carcinogenesis via activation of aryl hydrocarbon receptor signaling and consequent induction of oxidative DNA damage in rats.
Journal: Toxicology and applied pharmacology
Published: July 16, 2013
Showing 1-12 of 12
Last Updated: 09/26/2026