Tamaki Ishima

Shimotsuke, 3290498, Japan

Last Updated: 09/26/2026

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Found 67 publications

Proteomic Characterization of Replication Stress and Impaired Antioxidant Defense in Tacrolimus-Induced Chronic Nephrotoxicity.

Proteomic Characterization of Replication Stress and Impaired Antioxidant Defense in Tacrolimus-Induced Chronic Nephrotoxicity.

Journal: International journal of molecular sciences

Published: June 10, 2026

A Pathogenic ROCK-Signaling Network Involving a Lysine Deletion in Myh11 Renders Carriers Susceptible to Aortic Dissection.

A Pathogenic ROCK-Signaling Network Involving a Lysine Deletion in Myh11 Renders Carriers Susceptible to Aortic Dissection.

Journal: International journal of molecular sciences

Published: February 13, 2026

Trans-Omic Analysis Identifies the 'PRMT1-STAT3-Integrin αVβ6 Axis' as a Novel Therapeutic Target in Tacrolimus-Induced Chronic Nephrotoxicity.

Trans-Omic Analysis Identifies the 'PRMT1-STAT3-Integrin αVβ6 Axis' as a Novel Therapeutic Target in Tacrolimus-Induced Chronic Nephrotoxicity.

Journal: International journal of molecular sciences

Published: August 30, 2025

Targeting the undruggable transcription factor, KLF5, with a peptidomimetic small molecule, NC114, attenuates pressure overload-induced cardiac remodeling and fibrosis.

Targeting the undruggable transcription factor, KLF5, with a peptidomimetic small molecule, NC114, attenuates pressure overload-induced cardiac remodeling and fibrosis.

Journal: Scientific reports

Published: August 15, 2025

DHA-Triacylglycerol Accumulation in Tacrolimus-Induced Nephrotoxicity Identified by Lipidomic Profiling.

DHA-Triacylglycerol Accumulation in Tacrolimus-Induced Nephrotoxicity Identified by Lipidomic Profiling.

Journal: International journal of molecular sciences

Published: July 09, 2025

Loss of Myh11 K1256 Dysregulates the Extracellular Matrix and Focal Adhesion by Inhibiting Zyxin-Activated Transcription.

Loss of Myh11 K1256 Dysregulates the Extracellular Matrix and Focal Adhesion by Inhibiting Zyxin-Activated Transcription.

Journal: International journal of molecular sciences

Published: May 29, 2025

A Highly Potent Apomorphine Derivative Enhancing Neurite Outgrowth via Nrf2 Activation.

A Highly Potent Apomorphine Derivative Enhancing Neurite Outgrowth via Nrf2 Activation.

Journal: Antioxidants (Basel, Switzerland)

Published: February 26, 2025

Whole Blood Metabolomic Profiling of Mice with Tacrolimus-Induced Chronic Nephrotoxicity: NAD+ Depletion with Salvage Pathway Impairment.

Whole Blood Metabolomic Profiling of Mice with Tacrolimus-Induced Chronic Nephrotoxicity: NAD+ Depletion with Salvage Pathway Impairment.

Journal: Antioxidants (Basel, Switzerland)

Published: December 05, 2024

NADH Reductive Stress and Its Correlation with Disease Severity in Leigh Syndrome: A Pilot Study Using Patient Fibroblasts and a Mouse Model.

NADH Reductive Stress and Its Correlation with Disease Severity in Leigh Syndrome: A Pilot Study Using Patient Fibroblasts and a Mouse Model.

Journal: Biomolecules

Published: November 20, 2024

T-Type Voltage-Gated Calcium Channels: Potential Regulators of Smooth Muscle Contractility.

T-Type Voltage-Gated Calcium Channels: Potential Regulators of Smooth Muscle Contractility.

Journal: International journal of molecular sciences

Published: October 27, 2024

A Simple, Fast, Sensitive LC-MS/MS Method to Quantify NAD(H) in Biological Samples: Plasma NAD(H) Measurement to Monitor Brain Pathophysiology.

A Simple, Fast, Sensitive LC-MS/MS Method to Quantify NAD(H) in Biological Samples: Plasma NAD(H) Measurement to Monitor Brain Pathophysiology.

Journal: International journal of molecular sciences

Published: January 19, 2024

Metabolomic Profiling of Mice with Tacrolimus-Induced Nephrotoxicity: Carnitine Deficiency in Renal Tissue.

Metabolomic Profiling of Mice with Tacrolimus-Induced Nephrotoxicity: Carnitine Deficiency in Renal Tissue.

Journal: Biomedicines

Published: December 28, 2023

Showing 1-12 of 67

Last Updated: 09/26/2026