Tania Rizo

Erlangen, BY, Germany

Last Updated: 09/26/2026

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Found 8 publications

TNF alpha unmasks enteric malate aspartate shuttle dysfunction bridging Parkinson disease and intestinal inflammation.

TNF alpha unmasks enteric malate aspartate shuttle dysfunction bridging Parkinson disease and intestinal inflammation.

Journal: Nature communications
Published: May 28, 2025

TNF-α disrupts the malate-aspartate shuttle, driving metabolic rewiring in iPSC-derived enteric neural lineages from Parkinson's Disease patients.

TNF-α disrupts the malate-aspartate shuttle, driving metabolic rewiring in iPSC-derived enteric neural lineages from Parkinson's Disease patients.

Journal: bioRxiv : the preprint server for biology
Published: April 08, 2025

Recombinant pro-CTSD (cathepsin D) enhances SNCA/α-Synuclein degradation in α-Synucleinopathy models.

Recombinant pro-CTSD (cathepsin D) enhances SNCA/α-Synuclein degradation in α-Synucleinopathy models.

Journal: Autophagy
Published: March 15, 2022

Store-operated calcium entry is reduced in spastin-linked hereditary spastic paraplegia.

Store-operated calcium entry is reduced in spastin-linked hereditary spastic paraplegia.

Journal: Brain : a journal of neurology
Published: January 12, 2021

Modeling Cell-Cell Interactions in Parkinson's Disease Using Human Stem Cell-Based Models.

Modeling Cell-Cell Interactions in Parkinson's Disease Using Human Stem Cell-Based Models.

Journal: Frontiers in cellular neuroscience
Published: September 13, 2019

Mechanistic basis of an epistatic interaction reducing age at onset in hereditary spastic paraplegia.

Mechanistic basis of an epistatic interaction reducing age at onset in hereditary spastic paraplegia.

Journal: Brain : a journal of neurology
Published: August 22, 2017

Defects in ER-endosome contacts impact lysosome function in hereditary spastic paraplegia.

Defects in ER-endosome contacts impact lysosome function in hereditary spastic paraplegia.

Journal: The Journal of cell biology
Published: September 07, 2016

NLRP3 inflammasome is expressed by astrocytes in the SOD1 mouse model of ALS and in human sporadic ALS patients.

NLRP3 inflammasome is expressed by astrocytes in the SOD1 mouse model of ALS and in human sporadic ALS patients.

Journal: Glia
Published: March 10, 2015
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Last Updated: 09/26/2026