Tarik Regad

Clifton Lane
Nottingham, ENG, United Kingdom

Last Updated: 09/26/2026

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Found 24 publications

Correction: MiR-423-5p is a metabolic and growth tuner in hepatocellular carcinoma via MALAT-1 and mitochondrial interaction.

Correction: MiR-423-5p is a metabolic and growth tuner in hepatocellular carcinoma via MALAT-1 and mitochondrial interaction.

Journal: Journal of experimental & clinical cancer research : CR

Published: November 06, 2025

MiR-423-5p is a metabolic and growth tuner in hepatocellular carcinoma via MALAT-1 and mitochondrial interaction.

MiR-423-5p is a metabolic and growth tuner in hepatocellular carcinoma via MALAT-1 and mitochondrial interaction.

Journal: Journal of experimental & clinical cancer research : CR

Published: July 23, 2025

A Proteomic Approach Reveals That miR-423-5p Modulates Glucidic and Amino Acid Metabolism in Prostate Cancer Cells.

A Proteomic Approach Reveals That miR-423-5p Modulates Glucidic and Amino Acid Metabolism in Prostate Cancer Cells.

Journal: International journal of molecular sciences

Published: November 18, 2022

MiR-423-5p prevents MALAT1-mediated proliferation and metastasis in prostate cancer.

MiR-423-5p prevents MALAT1-mediated proliferation and metastasis in prostate cancer.

Journal: Journal of experimental & clinical cancer research : CR

Published: July 26, 2021

Long non-coding RNAs in cancer stem cells.

Long non-coding RNAs in cancer stem cells.

Journal: Translational oncology

Published: November 04, 2020

β2-AR blockade potentiates MEK1/2 inhibitor effect on HNSCC by regulating the Nrf2-mediated defense mechanism.

β2-AR blockade potentiates MEK1/2 inhibitor effect on HNSCC by regulating the Nrf2-mediated defense mechanism.

Journal: Cell death & disease

Published: May 20, 2020

Correction: MTSS1 and SCAMP1 cooperate to prevent invasion in breast cancer.

Correction: MTSS1 and SCAMP1 cooperate to prevent invasion in breast cancer.

Journal: Cell death & disease

Published: March 25, 2020

Cancer Stem Cells and Targeting Strategies.

Cancer Stem Cells and Targeting Strategies.

Journal: Cells

Published: July 05, 2019

Glucose-6-phosphate dehydrogenase blockade potentiates tyrosine kinase inhibitor effect on breast cancer cells through autophagy perturbation.

Glucose-6-phosphate dehydrogenase blockade potentiates tyrosine kinase inhibitor effect on breast cancer cells through autophagy perturbation.

Journal: Journal of experimental & clinical cancer research : CR

Published: February 07, 2019

PYK2 promotes HER2-positive breast cancer invasion.

PYK2 promotes HER2-positive breast cancer invasion.

Journal: Journal of experimental & clinical cancer research : CR

Published: January 25, 2019

HDAC2 depletion promotes osteosarcoma's stemness both in vitro and in vivo: a study on a putative new target for CSCs directed therapy.

HDAC2 depletion promotes osteosarcoma's stemness both in vitro and in vivo: a study on a putative new target for CSCs directed therapy.

Journal: Journal of experimental & clinical cancer research : CR

Published: October 02, 2018

A new inhibitor of glucose-6-phosphate dehydrogenase blocks pentose phosphate pathway and suppresses malignant proliferation and metastasis in vivo.

A new inhibitor of glucose-6-phosphate dehydrogenase blocks pentose phosphate pathway and suppresses malignant proliferation and metastasis in vivo.

Journal: Cell death & disease

Published: March 23, 2018

Showing 1-12 of 24

Last Updated: 09/26/2026