Tatyana Ponomaryov

Stevenage, ENG, United Kingdom

Last Updated: 09/26/2026

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Found 9 publications

From iPSC to manufactured iNK cells using CombiCult® screening platform.

From iPSC to manufactured iNK cells using CombiCult® screening platform.

Journal: Frontiers in cell and developmental biology

Published: March 05, 2026

Human iPSC-derived platelets generated in alginate scaffolds exhibit functional haemostasis in vitro and in vivo.

Human iPSC-derived platelets generated in alginate scaffolds exhibit functional haemostasis in vitro and in vivo.

Journal: Thrombosis research

Published: May 30, 2025

Neutrophil extracellular traps and inflammasomes cooperatively promote venous thrombosis in mice.

Neutrophil extracellular traps and inflammasomes cooperatively promote venous thrombosis in mice.

Journal: Blood advances

Published: September 09, 2020

Corrigendum: Large-scale production of megakaryocytes from human pluripotent stem cells by chemically defined forward programming.

Corrigendum: Large-scale production of megakaryocytes from human pluripotent stem cells by chemically defined forward programming.

Journal: Nature communications

Published: July 29, 2017

Mast Cells Granular Contents Are Crucial for Deep Vein Thrombosis in Mice.

Mast Cells Granular Contents Are Crucial for Deep Vein Thrombosis in Mice.

Journal: Circulation research

Published: April 16, 2017

A Transposon Screen Identifies Loss of Primary Cilia as a Mechanism of Resistance to SMO Inhibitors.

A Transposon Screen Identifies Loss of Primary Cilia as a Mechanism of Resistance to SMO Inhibitors.

Journal: Cancer discovery

Published: March 16, 2017

Mice with a deficiency in CLEC-2 are protected against deep vein thrombosis.

Mice with a deficiency in CLEC-2 are protected against deep vein thrombosis.

Journal: Blood

Published: September 28, 2016

Large-scale production of megakaryocytes from human pluripotent stem cells by chemically defined forward programming.

Large-scale production of megakaryocytes from human pluripotent stem cells by chemically defined forward programming.

Journal: Nature communications

Published: June 23, 2015

RAS/MAPK Activation Drives Resistance to Smo Inhibition, Metastasis, and Tumor Evolution in Shh Pathway-Dependent Tumors.

RAS/MAPK Activation Drives Resistance to Smo Inhibition, Metastasis, and Tumor Evolution in Shh Pathway-Dependent Tumors.

Journal: Cancer research

Published: October 20, 2014

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Last Updated: 09/26/2026