Teresa Garcia-Crespo
Madrid, MD, Spain
Last Updated: 09/26/2026
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Found 9 publications
Propionic Acidemia-Induced Proarrhythmic Electrophysiological Alterations in Human iPSC-Derived Cardiomyocytes.
Journal: Journal of inherited metabolic disease
Published: January 08, 2025
Two concurrent mechanisms are responsible for the INa increase produced by dapagliflozin and empagliflozin in healthy and heart failure cardiomyocytes.
Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
Published: December 19, 2024
Reply to Benndorff and DiFrancesco: Reliable human HCN4 single-channel recordings using the cell-attached configuration in expression systems.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: April 08, 2024
A gain-of-function HCN4 mutant in the HCN domain is responsible for inappropriate sinus tachycardia in a Spanish family.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: November 30, 2023
Empagliflozin and Dapagliflozin Increase Na+ and Inward Rectifier K+ Current Densities in Human Cardiomyocytes Derived from Induced Pluripotent Stem Cells (hiPSC-CMs).
Journal: Cells
Published: September 29, 2022
Regulation of cardiac ion channels by transcription factors: Looking for new opportunities of druggable targets for the treatment of arrhythmias.
Journal: Biochemical pharmacology
Published: June 27, 2022
Zfhx3 Transcription Factor Represses the Expression of SCN5A Gene and Decreases Sodium Current Density (INa).
Journal: International journal of molecular sciences
Published: November 02, 2021
Tbx5 variants disrupt Nav1.5 function differently in patients diagnosed with Brugada or Long QT Syndrome.
Journal: Cardiovascular research
Published: October 06, 2020
The p.P888L SAP97 polymorphism increases the transient outward current (Ito,f) and abbreviates the action potential duration and the QT interval.
Journal: Scientific reports
Published: December 26, 2019
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Last Updated: 09/26/2026