Thomas A. Blair

Bristol, ENG BS81TD, United Kingdom

Last Updated: 09/26/2026

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Found 8 publications

Rapamycin restrains platelet procoagulant responses via FKBP-mediated protection of mitochondrial integrity.

Rapamycin restrains platelet procoagulant responses via FKBP-mediated protection of mitochondrial integrity.

Journal: Biochemical pharmacology

Published: January 27, 2020

Platelet surface marker analysis by mass cytometry.

Platelet surface marker analysis by mass cytometry.

Journal: Platelets

Published: September 24, 2019

Critical roles for the phosphatidylinositide 3-kinase isoforms p110β and p110γ in thrombopoietin-mediated priming of platelet function.

Critical roles for the phosphatidylinositide 3-kinase isoforms p110β and p110γ in thrombopoietin-mediated priming of platelet function.

Journal: Scientific reports

Published: June 22, 2018

Mass Cytometry Reveals Distinct Platelet Subtypes in Healthy Subjects and Novel Alterations in Surface Glycoproteins in Glanzmann Thrombasthenia.

Mass Cytometry Reveals Distinct Platelet Subtypes in Healthy Subjects and Novel Alterations in Surface Glycoproteins in Glanzmann Thrombasthenia.

Journal: Scientific reports

Published: April 12, 2018

Phosphoinositide 3-kinase p110α negatively regulates thrombopoietin-mediated platelet activation and thrombus formation.

Phosphoinositide 3-kinase p110α negatively regulates thrombopoietin-mediated platelet activation and thrombus formation.

Journal: Cellular signalling

Published: February 06, 2018

Circulating primers enhance platelet function and induce resistance to antiplatelet therapy.

Circulating primers enhance platelet function and induce resistance to antiplatelet therapy.

Journal: Journal of thrombosis and haemostasis : JTH

Published: March 22, 2015

Loss of the insulin receptor in murine megakaryocytes/platelets causes thrombocytosis and alterations in IGF signalling.

Loss of the insulin receptor in murine megakaryocytes/platelets causes thrombocytosis and alterations in IGF signalling.

Journal: Cardiovascular research

Published: July 25, 2014

Phosphoinositide 3-kinases p110α and p110β have differential roles in insulin-like growth factor-1-mediated Akt phosphorylation and platelet priming.

Phosphoinositide 3-kinases p110α and p110β have differential roles in insulin-like growth factor-1-mediated Akt phosphorylation and platelet priming.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: June 07, 2014

Showing 1-8 of 8

Last Updated: 09/26/2026