Thomas Weide
Muenster, NW, Germany
Last Updated: 09/26/2026
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Found 39 publications
APOL1 plasma membrane pools resist rapid protein degradation.
Journal: Scientific reports
Published: November 17, 2025
The APOL1 variant p.N264K is predicted to block ion flow by occluding a pore at the cell surface.
Journal: Life science alliance
Published: November 17, 2025
Mechanisms of Podocyte Injury Due to Loss of the Nucleoporin NUP93.
Journal: Journal of the American Society of Nephrology : JASN
Published: June 19, 2025
A Cell Biologist's View on APOL1: What We Know and What We Still Need to Address.
Journal: Cells
Published: May 15, 2025
Crumbs2 acts as a dynamic cellular sensor triggering stable podocyte cell-matrix anchorage.
Journal: Biophysical journal
Published: January 28, 2025
PATJ inhibits histone deacetylase 7 to control tight junction formation and cell polarity.
Journal: Cellular and molecular life sciences : CMLS
Published: June 06, 2023
PALS1 is a key regulator of the lateral distribution of tight junction proteins in renal epithelial cells.
Journal: Journal of cell science
Published: May 03, 2023
Disruption of the Rab7-Dependent Final Common Pathway of Endosomal and Autophagic Processing Results in a Severe Podocytopathy.
Journal: Journal of the American Society of Nephrology : JASN
Published: October 26, 2022
Loss of surface transport is a main cellular pathomechanism of CRB2 variants causing podocytopathies.
Journal: Life science alliance
Published: August 04, 2022
Last Updated: 09/26/2026