Tianhui Wang

333 Nanchen Road
Baoshan, 200444, China

Last Updated: 09/26/2026

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Found 18 publications

YTHDF1-mediated m6A RNA regulation controls cardiomyocyte remodeling through KLF11-HIF1α.

YTHDF1-mediated m6A RNA regulation controls cardiomyocyte remodeling through KLF11-HIF1α.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: September 01, 2026

EV-Mediated Intracardiac Crosstalk Mitigates Doxorubicin-Induced Cardiotoxicity.

EV-Mediated Intracardiac Crosstalk Mitigates Doxorubicin-Induced Cardiotoxicity.

Journal: Circulation research

Published: April 08, 2026

Targeting Mettl14 Using an RNA-Targeting Clustered Regularly Interspaced Short Palindromic Repeat-High-Fidelity Cas13x System Attenuates Doxorubicin-Induced Cardiotoxicity.

Targeting Mettl14 Using an RNA-Targeting Clustered Regularly Interspaced Short Palindromic Repeat-High-Fidelity Cas13x System Attenuates Doxorubicin-Induced Cardiotoxicity.

Journal: Journal of the American Heart Association

Published: July 17, 2025

Cascade Nanozyme Delivering miRNA to Ischemic Heart to Alleviate Myocardial Ischemia-Reperfusion Injury.

Cascade Nanozyme Delivering miRNA to Ischemic Heart to Alleviate Myocardial Ischemia-Reperfusion Injury.

Journal: Small (Weinheim an der Bergstrasse, Germany)

Published: March 03, 2025

Nanoparticle-Patch System for Localized, Effective, and Sustained miRNA Administration into Infarcted Myocardium to Alleviate Myocardial Ischemia-Reperfusion Injury.

Nanoparticle-Patch System for Localized, Effective, and Sustained miRNA Administration into Infarcted Myocardium to Alleviate Myocardial Ischemia-Reperfusion Injury.

Journal: ACS nano

Published: July 17, 2024

MiR-421 mediates PM2.5-induced endothelial dysfunction via crosstalk between bronchial epithelial and endothelial cells.

MiR-421 mediates PM2.5-induced endothelial dysfunction via crosstalk between bronchial epithelial and endothelial cells.

Journal: Inhalation toxicology

Published: May 22, 2024

Exercise Inhibits Doxorubicin-Induced Cardiotoxicity via Regulating B Cells.

Exercise Inhibits Doxorubicin-Induced Cardiotoxicity via Regulating B Cells.

Journal: Circulation research

Published: February 07, 2024

Exercise training decreases lactylation and prevents myocardial ischemia-reperfusion injury by inhibiting YTHDF2.

Exercise training decreases lactylation and prevents myocardial ischemia-reperfusion injury by inhibiting YTHDF2.

Journal: Basic research in cardiology

Published: August 12, 2023

Exercise-induced circular RNA circUtrn is required for cardiac physiological hypertrophy and prevents myocardial ischaemia-reperfusion injury.

Exercise-induced circular RNA circUtrn is required for cardiac physiological hypertrophy and prevents myocardial ischaemia-reperfusion injury.

Journal: Cardiovascular research

Published: January 06, 2023

RNA m6A-Regulated circ-ZNF609 Suppression Ameliorates Doxorubicin-Induced Cardiotoxicity by Upregulating FTO.

RNA m6A-Regulated circ-ZNF609 Suppression Ameliorates Doxorubicin-Induced Cardiotoxicity by Upregulating FTO.

Journal: JACC. Basic to translational science

Published: September 23, 2022

Downregulation of circ-ZNF609 Promotes Heart Repair by Modulating RNA N6-Methyladenosine-Modified Yap Expression.

Downregulation of circ-ZNF609 Promotes Heart Repair by Modulating RNA N6-Methyladenosine-Modified Yap Expression.

Journal: Research (Washington, D.C.)

Published: November 24, 2021

Extracellular vesicles enclosed-miR-421 suppresses air pollution (PM2.5 )-induced cardiac dysfunction via ACE2 signalling.

Extracellular vesicles enclosed-miR-421 suppresses air pollution (PM2.5 )-induced cardiac dysfunction via ACE2 signalling.

Journal: Journal of extracellular vesicles

Published: October 14, 2021

Showing 1-12 of 18

Last Updated: 09/26/2026