Tomohiko Irie

Sagamihara, Japan

Last Updated: 09/26/2026

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Found 20 publications

The Na+ leak channel NALCN controls spontaneous activity and mediates synaptic modulation by α2-adrenergic receptors in auditory neurons.

The Na+ leak channel NALCN controls spontaneous activity and mediates synaptic modulation by α2-adrenergic receptors in auditory neurons.

Journal: eLife

Published: January 10, 2024

The Na+ leak channel NALCN controls spontaneous activity and mediates synaptic modulation by α2-adrenergic receptors in auditory neurons.

The Na+ leak channel NALCN controls spontaneous activity and mediates synaptic modulation by α2-adrenergic receptors in auditory neurons.

Journal: bioRxiv : the preprint server for biology

Published: November 21, 2023

Correction: Leucine 434 is essential for docosahexaenoic acid-induced augmentation of L-glutamate transporter current.

Correction: Leucine 434 is essential for docosahexaenoic acid-induced augmentation of L-glutamate transporter current.

Journal: The Journal of biological chemistry

Published: June 22, 2023

Derivatives of methoxetamine and major methoxetamine metabolites potently block NMDA receptors.

Derivatives of methoxetamine and major methoxetamine metabolites potently block NMDA receptors.

Journal: Journal of pharmacological sciences

Published: April 17, 2022

Leucine 434 is essential for docosahexaenoic acid-induced augmentation of L-glutamate transporter current.

Leucine 434 is essential for docosahexaenoic acid-induced augmentation of L-glutamate transporter current.

Journal: The Journal of biological chemistry

Published: April 13, 2022

Valproic acid promotes differentiation of adipose tissue-derived stem cells to neuronal cells selectively expressing functional N-type voltage-gated Ca2+ channels.

Valproic acid promotes differentiation of adipose tissue-derived stem cells to neuronal cells selectively expressing functional N-type voltage-gated Ca2+ channels.

Journal: Biochemical and biophysical research communications

Published: October 15, 2021

Essential Role of Somatic Kv2 Channels in High-Frequency Firing in Cartwheel Cells of the Dorsal Cochlear Nucleus.

Essential Role of Somatic Kv2 Channels in High-Frequency Firing in Cartwheel Cells of the Dorsal Cochlear Nucleus.

Journal: eNeuro

Published: November 30, 2020

F-phenibut (β-(4-Fluorophenyl)-GABA), a potent GABAB receptor agonist, activates an outward-rectifying K+ current and suppresses the generation of action potentials in mouse cerebellar Purkinje cells.

F-phenibut (β-(4-Fluorophenyl)-GABA), a potent GABAB receptor agonist, activates an outward-rectifying K+ current and suppresses the generation of action potentials in mouse cerebellar Purkinje cells.

Journal: European journal of pharmacology

Published: March 31, 2020

Inhibitory and inductive effects of 4- or 5-methyl-2-mercaptobenzimidazole, thyrotoxic and hepatotoxic rubber antioxidants, on several forms of cytochrome P450 in primary cultured rat and human hepatocytes.

Inhibitory and inductive effects of 4- or 5-methyl-2-mercaptobenzimidazole, thyrotoxic and hepatotoxic rubber antioxidants, on several forms of cytochrome P450 in primary cultured rat and human hepatocytes.

Journal: Toxicology reports

Published: January 17, 2020

Loose coupling between SK and P/Q-type Ca2+ channels in cartwheel cells of the dorsal cochlear nucleus.

Loose coupling between SK and P/Q-type Ca2+ channels in cartwheel cells of the dorsal cochlear nucleus.

Journal: Journal of neurophysiology

Published: August 29, 2019

Sub-toxic concentrations of nano-ZnO and nano-TiO2 suppress neurite outgrowth in differentiated PC12 cells.

Sub-toxic concentrations of nano-ZnO and nano-TiO2 suppress neurite outgrowth in differentiated PC12 cells.

Journal: The Journal of toxicological sciences

Published: November 17, 2017

Thyrotoxic rubber antioxidants, 2-mercaptobenzimidazole and its methyl derivatives, cause both inhibition and induction of drug-metabolizing activity in rat liver microsomes after repeated oral administration.

Thyrotoxic rubber antioxidants, 2-mercaptobenzimidazole and its methyl derivatives, cause both inhibition and induction of drug-metabolizing activity in rat liver microsomes after repeated oral administration.

Journal: Biochemical and biophysical research communications

Published: July 27, 2017

Showing 1-12 of 20

Last Updated: 09/26/2026