Toshihisa Hatta

Kahoku, Japan

Last Updated: 09/26/2026

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Found 36 publications

CaOx crystal nuclei are formed in rat outer cortex proximal tubules by a potential fibrinogen-dependent mechanism.

CaOx crystal nuclei are formed in rat outer cortex proximal tubules by a potential fibrinogen-dependent mechanism.

Journal: PloS one

Published: November 29, 2024

Cellular automata modelling of leukaemic stem cell dynamics in acute myeloid leukaemia: insights into predictive outcomes and targeted therapies.

Cellular automata modelling of leukaemic stem cell dynamics in acute myeloid leukaemia: insights into predictive outcomes and targeted therapies.

Journal: Royal Society open science

Published: July 23, 2024

Histological changes in the olfactory bulb and rostral migratory stream due to interruption of olfactory input.

Histological changes in the olfactory bulb and rostral migratory stream due to interruption of olfactory input.

Journal: Auris, nasus, larynx

Published: October 23, 2023

Spatiotemporal development of the neuronal accumulation of amyloid precursor protein and the amyloid plaque formation in the brain of 3xTg-AD mice.

Spatiotemporal development of the neuronal accumulation of amyloid precursor protein and the amyloid plaque formation in the brain of 3xTg-AD mice.

Journal: Heliyon

Published: August 28, 2023

Glycolytic activity is required for the onset of neural plate folding during neural tube closure in mouse embryos.

Glycolytic activity is required for the onset of neural plate folding during neural tube closure in mouse embryos.

Journal: Frontiers in cell and developmental biology

Published: April 26, 2023

Influence of Immune System Abnormalities Caused by Maternal Immune Activation in the Postnatal Period.

Influence of Immune System Abnormalities Caused by Maternal Immune Activation in the Postnatal Period.

Journal: Cells

Published: January 11, 2023

LIF-IGF Axis Contributes to the Proliferation of Neural Progenitor Cells in Developing Rat Cerebrum.

LIF-IGF Axis Contributes to the Proliferation of Neural Progenitor Cells in Developing Rat Cerebrum.

Journal: International journal of molecular sciences

Published: May 07, 2022

Hif1α-dependent hypoxia signaling contributes to the survival of deep-layer neurons and cortex formation in a mouse model.

Hif1α-dependent hypoxia signaling contributes to the survival of deep-layer neurons and cortex formation in a mouse model.

Journal: Molecular brain

Published: September 27, 2021

Maternal Exercise-Induced SOD3 Reverses the Deleterious Effects of Maternal High-Fat Diet on Offspring Metabolism Through Stabilization of H3K4me3 and Protection Against WDR82 Carbonylation.

Maternal Exercise-Induced SOD3 Reverses the Deleterious Effects of Maternal High-Fat Diet on Offspring Metabolism Through Stabilization of H3K4me3 and Protection Against WDR82 Carbonylation.

Journal: Diabetes

Published: August 09, 2021

Cerebrospinal fluid may flow out from the brain through the frontal skull base and choroid plexus: a gold colloid and cadaverine injection study in mouse fetus.

Cerebrospinal fluid may flow out from the brain through the frontal skull base and choroid plexus: a gold colloid and cadaverine injection study in mouse fetus.

Journal: Child's nervous system : ChNS : official journal of the International Society for Pediatric Neurosurgery

Published: March 18, 2021

Mid-pregnancy maternal immune activation increases Pax6-positive and Tbr2-positive neural progenitor cells and causes integrated stress response in the fetal brain in a mouse model of maternal viral infection.

Mid-pregnancy maternal immune activation increases Pax6-positive and Tbr2-positive neural progenitor cells and causes integrated stress response in the fetal brain in a mouse model of maternal viral infection.

Journal: IBRO neuroscience reports

Published: March 08, 2021

Exposure to Maternal Immune Activation Causes Congenital Unfolded Protein Response Defects and Increases the Susceptibility to Postnatal Inflammatory Stimulation in Offspring.

Exposure to Maternal Immune Activation Causes Congenital Unfolded Protein Response Defects and Increases the Susceptibility to Postnatal Inflammatory Stimulation in Offspring.

Journal: Journal of inflammation research

Published: December 01, 2020

Showing 1-12 of 36

Last Updated: 09/26/2026