Ulrike C. Muller
Heidelberg, BW, Germany
Last Updated: 09/26/2026
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Found 93 publications
Endothelial soluble APP/APLP2 promote heart repair through KIT-mediated angiogenesis.
Journal: Science advances
Published: May 22, 2026
Brain delivery of a neurotrophic peptide derived from secreted amyloid precursor protein APPsα as a therapeutic strategy for Alzheimer's disease.
Journal: Journal of controlled release : official journal of the Controlled Release Society
Published: June 30, 2025
Trans-dimerization of Amyloid Precursor Protein Family Members Induces Pre- and Postsynaptic Differentiation through Distinct Signaling Pathways.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: April 08, 2025
The amyloid precursor family of proteins in excitatory neurons are essential for regulating cortico-hippocampal circuit dynamics in vivo.
Journal: Cell reports
Published: January 12, 2025
Taper wear in total joint arthroplasty can be reliably assessed with various coordinate measuring systems.
Journal: Scientific reports
Published: August 01, 2024
Author Correction: Aplp1 interacts with Lag3 to facilitate transmission of pathologic α-synuclein.
Journal: Nature communications
Published: July 30, 2024
CNTN4 modulates neural elongation through interplay with APP.
Journal: Open biology
Published: May 15, 2024
Consequences of Amyloid-β Deficiency for the Liver.
Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: October 15, 2023
Aplp1 interacts with Lag3 to facilitate transmission of pathologic α-synuclein.
Journal: Nature communications
Published: October 04, 2023
Axonal transport of Frizzled5 by Alcadein α-containing vesicles is associated with kinesin-1.
Journal: Molecular biology of the cell
Published: August 16, 2023
APP family member dimeric complexes are formed predominantly in synaptic compartments.
Journal: Cell & bioscience
Published: March 03, 2023
APPsα rescues CDK5 and GSK3β dysregulation and restores normal spine density in Tau transgenic mice.
APPsα rescues CDK5 and GSK3β dysregulation and restores normal spine density in Tau transgenic mice.
Journal: Frontiers in cellular neuroscience
Published: November 23, 2022
Last Updated: 09/26/2026