Veerle Paternoster
Arhus, Denmark
Last Updated: 09/26/2026
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Found 8 publications
The psychiatric risk gene BRD1 modulates mitochondrial bioenergetics by transcriptional regulation.
Journal: Translational psychiatry
Published: January 13, 2022
Inactivation of the Schizophrenia-associated BRD1 gene in Brain Causes Failure-to-thrive, Seizure Susceptibility and Abnormal Histone H3 Acetylation and N-tail Clipping.
Journal: Molecular neurobiology
Published: February 11, 2021
Reduced Brd1 expression leads to reversible depression-like behaviors and gene-expression changes in female mice.
Journal: Translational psychiatry
Published: February 03, 2020
Brain volumetric alterations accompanied with loss of striatal medium-sized spiny neurons and cortical parvalbumin expressing interneurons in Brd1+/- mice.
Journal: Scientific reports
Published: June 07, 2018
Brain proteome changes in female Brd1+/- mice unmask dendritic spine pathology and show enrichment for schizophrenia risk.
Journal: Neurobiology of disease
Published: April 25, 2018
The importance of data structure in statistical analysis of dendritic spine morphology.
Journal: Journal of neuroscience methods
Published: September 19, 2017
The Schizophrenia-Associated BRD1 Gene Regulates Behavior, Neurotransmission, and Expression of Schizophrenia Risk Enriched Gene Sets in Mice.
Journal: Biological psychiatry
Published: February 05, 2016
Quantitative assessment of methyl-esterification and other side reactions in a standard propionylation protocol for detection of histone modifications.
Journal: Proteomics
Published: November 07, 2015
Showing 1-8 of 8
Last Updated: 09/26/2026