Vera I. Wiersma
Zurich, ZH, Switzerland
Last Updated: 09/26/2026
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Found 11 publications
Granulovacuolar degeneration bodies are independently induced by tau and α-synuclein pathology.
Journal: Alzheimer's research & therapy
Published: August 16, 2022
Loss of TDP-43 oligomerization or RNA binding elicits distinct aggregation patterns.
Journal: The EMBO journal
Published: May 20, 2022
Sharing is caring: The benefits of distributing protein aggregates among microglial networks.
Journal: Neuron
Published: October 21, 2021
A model of human neural networks reveals NPTX2 pathology in ALS and FTLD.
Journal: Nature
Published: September 28, 2021
FTLD-TDP assemblies seed neoaggregates with subtype-specific features via a prion-like cascade.
Journal: EMBO reports
Published: August 25, 2021
Untangling the origin and function of granulovacuolar degeneration bodies in neurodegenerative proteinopathies.
Journal: Acta neuropathologica communications
Published: July 14, 2020
Granulovacuolar degeneration bodies: red alert for neurons with MAPT/tau pathology.
Journal: Autophagy
Published: October 16, 2019
Activation of the unfolded protein response and granulovacuolar degeneration are not common features of human prion pathology.
Journal: Acta neuropathologica communications
Published: October 05, 2016
Showing 1-11 of 11
Last Updated: 09/26/2026