Vera Lemos

Lausanne, VD 1015, Switzerland

Last Updated: 09/26/2026

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Found 8 publications

The Slc25a47 locus is a novel determinant of hepatic mitochondrial function implicated in liver fibrosis.

The Slc25a47 locus is a novel determinant of hepatic mitochondrial function implicated in liver fibrosis.

Journal: Journal of hepatology

Published: November 26, 2021

A new class of protein biomarkers based on subcellular distribution: application to a mouse liver cancer model.

A new class of protein biomarkers based on subcellular distribution: application to a mouse liver cancer model.

Journal: Scientific reports

Published: February 27, 2019

The RNA-Binding Protein PUM2 Impairs Mitochondrial Dynamics and Mitophagy During Aging.

The RNA-Binding Protein PUM2 Impairs Mitochondrial Dynamics and Mitophagy During Aging.

Journal: Molecular cell

Published: May 15, 2018

Fas cell surface death receptor controls hepatic lipid metabolism by regulating mitochondrial function.

Fas cell surface death receptor controls hepatic lipid metabolism by regulating mitochondrial function.

Journal: Nature communications

Published: July 12, 2016

Inhibiting poly ADP-ribosylation increases fatty acid oxidation and protects against fatty liver disease.

Inhibiting poly ADP-ribosylation increases fatty acid oxidation and protects against fatty liver disease.

Journal: Journal of hepatology

Published: April 20, 2016

Impaired SUMOylation of nuclear receptor LRH-1 promotes nonalcoholic fatty liver disease.

Impaired SUMOylation of nuclear receptor LRH-1 promotes nonalcoholic fatty liver disease.

Journal: The Journal of clinical investigation

Published: November 11, 2015

Eliciting the mitochondrial unfolded protein response by nicotinamide adenine dinucleotide repletion reverses fatty liver disease in mice.

Eliciting the mitochondrial unfolded protein response by nicotinamide adenine dinucleotide repletion reverses fatty liver disease in mice.

Journal: Hepatology (Baltimore, Md.)

Published: March 18, 2015

SUMOylation-dependent LRH-1/PROX1 interaction promotes atherosclerosis by decreasing hepatic reverse cholesterol transport.

SUMOylation-dependent LRH-1/PROX1 interaction promotes atherosclerosis by decreasing hepatic reverse cholesterol transport.

Journal: Cell metabolism

Published: March 04, 2014

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Last Updated: 09/26/2026