Veronique Gire

Montpellier, France

Last Updated: 09/26/2026

Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 9 publications

Interaction of p53 with the Δ133p53α and Δ160p53α isoforms regulates p53 conformation and transcriptional activity.

Interaction of p53 with the Δ133p53α and Δ160p53α isoforms regulates p53 conformation and transcriptional activity.

Journal: Cell death & disease
Published: April 12, 2024

Mechanisms and Regulation of Cellular Senescence.

Mechanisms and Regulation of Cellular Senescence.

Journal: International journal of molecular sciences
Published: October 22, 2021

Reciprocal regulation of p21 and Chk1 controls the cyclin D1-RB pathway to mediate senescence onset after G2 arrest.

Reciprocal regulation of p21 and Chk1 controls the cyclin D1-RB pathway to mediate senescence onset after G2 arrest.

Journal: Journal of cell science
Published: July 12, 2021

Greatwall promotes cell transformation by hyperactivating AKT in human malignancies.

Greatwall promotes cell transformation by hyperactivating AKT in human malignancies.

Journal: eLife
Published: July 15, 2015

Senescence from G2 arrest, revisited.

Senescence from G2 arrest, revisited.

Journal: Cell cycle (Georgetown, Tex.)
Published: January 08, 2015

The p53 isoform Δ133p53β promotes cancer stem cell potential.

The p53 isoform Δ133p53β promotes cancer stem cell potential.

Journal: Stem cell reports
Published: July 07, 2014

Eroded human telomeres are more prone to remain uncapped and to trigger a G2 checkpoint response.

Eroded human telomeres are more prone to remain uncapped and to trigger a G2 checkpoint response.

Journal: Nucleic acids research
Published: November 30, 2012

Senescence: a telomeric limit to immortality or a cellular response to physiologic stresses?

Senescence: a telomeric limit to immortality or a cellular response to physiologic stresses?

Journal: Medecine sciences : M/S
Published: May 12, 2005

Dysfunctional telomeres at senescence signal cell cycle arrest via Chk2.

Dysfunctional telomeres at senescence signal cell cycle arrest via Chk2.

Journal: Cell cycle (Georgetown, Tex.)
Published: October 07, 2004
Showing 1-9 of 9

Last Updated: 09/26/2026