Victoria Margulets
St. Boniface Hospital Albrechtsen Research Centre
Winnipeg, MB R2H2H6, Canada
Winnipeg, MB R2H2H6, Canada
Last Updated: 09/26/2026
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Found 14 publications
IKKβ stabilizes Mitofusin 2 and suppresses doxorubicin cardiomyopathy.
Journal: Cardiovascular research
Published: December 29, 2022
Ellagic acid inhibits mitochondrial fission protein Drp-1 and cell proliferation in cancer.
Journal: Molecular and cellular biochemistry
Published: September 21, 2022
Proteasomal Degradation of TRAF2 Mediates Mitochondrial Dysfunction in Doxorubicin-Cardiomyopathy.
Journal: Circulation
Published: August 19, 2022
Modulating mitofusins to control mitochondrial function and signaling.
Journal: Nature communications
Published: April 29, 2021
A small-molecule allosteric inhibitor of BAX protects against doxorubicin-induced cardiomyopathy.
Journal: Nature cancer
Published: August 11, 2020
Impaired NF-κB signalling underlies cyclophilin D-mediated mitochondrial permeability transition pore opening in doxorubicin cardiomyopathy.
Journal: Cardiovascular research
Published: June 07, 2019
Reduction of myocardial ischaemia-reperfusion injury by inactivating oxidized phospholipids.
Journal: Cardiovascular research
Published: March 05, 2018
Ellagic acid antagonizes Bnip3-mediated mitochondrial injury and necrotic cell death of cardiac myocytes.
Journal: Free radical biology & medicine
Published: April 05, 2017
PDK2-mediated alternative splicing switches Bnip3 from cell death to cell survival.
Journal: The Journal of cell biology
Published: September 30, 2015
Bcl-2/adenovirus E1B 19-kDa interacting protein (BNip3) has a key role in the mitochondrial dysfunction induced by mutant huntingtin.
Journal: Human molecular genetics
Published: May 28, 2015
Bnip3 mediates doxorubicin-induced cardiac myocyte necrosis and mortality through changes in mitochondrial signaling.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: December 10, 2014
Last Updated: 09/26/2026