Vijay K. Alla

Muenster, NW, Germany

Last Updated: 09/26/2026

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Found 12 publications

Epigenetic control over the cell-intrinsic immune response antagonizes self-renewal in acute myeloid leukemia.

Epigenetic control over the cell-intrinsic immune response antagonizes self-renewal in acute myeloid leukemia.

Journal: Blood
Published: June 29, 2023

The neuropeptide receptor calcitonin receptor-like (CALCRL) is a potential therapeutic target in acute myeloid leukemia.

The neuropeptide receptor calcitonin receptor-like (CALCRL) is a potential therapeutic target in acute myeloid leukemia.

Journal: Leukemia
Published: March 11, 2019

RNA-Guided CRISPR-Cas9 System-Mediated Engineering of Acute Myeloid Leukemia Mutations.

RNA-Guided CRISPR-Cas9 System-Mediated Engineering of Acute Myeloid Leukemia Mutations.

Journal: Molecular therapy. Nucleic acids
Published: December 14, 2016

p73 and IGF1R Regulate Emergence of Aggressive Cancer Stem-like Features via miR-885-5p Control.

p73 and IGF1R Regulate Emergence of Aggressive Cancer Stem-like Features via miR-885-5p Control.

Journal: Cancer research
Published: May 06, 2015

Epigenetic factor EPC1 is a master regulator of DNA damage response by interacting with E2F1 to silence death and activate metastasis-related gene signatures.

Epigenetic factor EPC1 is a master regulator of DNA damage response by interacting with E2F1 to silence death and activate metastasis-related gene signatures.

Journal: Nucleic acids research
Published: March 13, 2015

A balancing act: orchestrating amino-truncated and full-length p73 variants as decisive factors in cancer progression.

A balancing act: orchestrating amino-truncated and full-length p73 variants as decisive factors in cancer progression.

Journal: Oncogene
Published: August 08, 2014

Eradication of metastatic melanoma through cooperative expression of RNA-based HDAC1 inhibitor and p73 by oncolytic adenovirus.

Eradication of metastatic melanoma through cooperative expression of RNA-based HDAC1 inhibitor and p73 by oncolytic adenovirus.

Journal: Oncotarget
Published: July 30, 2014

Association of RHAMM with E2F1 promotes tumour cell extravasation by transcriptional up-regulation of fibronectin.

Association of RHAMM with E2F1 promotes tumour cell extravasation by transcriptional up-regulation of fibronectin.

Journal: The Journal of pathology
Published: March 28, 2014

E2F1 promotes angiogenesis through the VEGF-C/VEGFR-3 axis in a feedback loop for cooperative induction of PDGF-B.

E2F1 promotes angiogenesis through the VEGF-C/VEGFR-3 axis in a feedback loop for cooperative induction of PDGF-B.

Journal: Journal of molecular cell biology
Published: September 10, 2013

DNp73 exerts function in metastasis initiation by disconnecting the inhibitory role of EPLIN on IGF1R-AKT/STAT3 signaling.

DNp73 exerts function in metastasis initiation by disconnecting the inhibitory role of EPLIN on IGF1R-AKT/STAT3 signaling.

Journal: Cancer cell
Published: March 12, 2013

E2F1 confers anticancer drug resistance by targeting ABC transporter family members and Bcl-2 via the p73/DNp73-miR-205 circuitry.

E2F1 confers anticancer drug resistance by targeting ABC transporter family members and Bcl-2 via the p73/DNp73-miR-205 circuitry.

Journal: Cell cycle (Georgetown, Tex.)
Published: August 09, 2012

The human Lgl polarity gene, Hugl-2, induces MET and suppresses Snail tumorigenesis.

The human Lgl polarity gene, Hugl-2, induces MET and suppresses Snail tumorigenesis.

Journal: Oncogene
Published: May 15, 2012
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Last Updated: 09/26/2026