Wanping Zhang

Key Laboratory For Biochemistry And Molecular Pharmacology Of Chongqing
Chongqing, China

Last Updated: 09/26/2026

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Found 11 publications

Discovery of novel N-(prop-2-yn-1-yl)-1,2,3,4-tetrahydronaphthalen-1-amine derivatives as MAO-B inhibitors for the treatment of Parkinson's disease.

Discovery of novel N-(prop-2-yn-1-yl)-1,2,3,4-tetrahydronaphthalen-1-amine derivatives as MAO-B inhibitors for the treatment of Parkinson's disease.

Journal: Journal of enzyme inhibition and medicinal chemistry

Published: January 08, 2026

O-GlcNAcylation-dependent liquid-liquid phase separation regulates the nuclear translocation of YAP to exacerbate vascular neointimal hyperplasia.

O-GlcNAcylation-dependent liquid-liquid phase separation regulates the nuclear translocation of YAP to exacerbate vascular neointimal hyperplasia.

Journal: Theranostics

Published: March 06, 2025

Inhibition of the calcineurin/forkhead box O1/fatty acid binding protein 4 pathway prevents SERCA2 dysfunction-induced foam cell formation and atherosclerosis.

Inhibition of the calcineurin/forkhead box O1/fatty acid binding protein 4 pathway prevents SERCA2 dysfunction-induced foam cell formation and atherosclerosis.

Journal: British journal of pharmacology

Published: March 03, 2025

Calcium dysregulation disrupts mitochondrial homeostasis by interfering AMPK/Drp1 pathway to aggravate plaque progression and instability.

Calcium dysregulation disrupts mitochondrial homeostasis by interfering AMPK/Drp1 pathway to aggravate plaque progression and instability.

Journal: Theranostics

Published: February 12, 2025

N-Acetylneuraminic acid triggers endothelial pyroptosis and promotes atherosclerosis progression via GLS2-mediated glutaminolysis pathway.

N-Acetylneuraminic acid triggers endothelial pyroptosis and promotes atherosclerosis progression via GLS2-mediated glutaminolysis pathway.

Journal: Cell death discovery

Published: June 27, 2024

REDD1 knockdown ameliorates endothelial cell senescence through repressing TXNIP-mediated oxidative stress.

REDD1 knockdown ameliorates endothelial cell senescence through repressing TXNIP-mediated oxidative stress.

Journal: Mechanisms of ageing and development

Published: May 11, 2024

Pillar[6]MaxQ functions as an in vivo sequestrant for rocuronium and vecuronium.

Pillar[6]MaxQ functions as an in vivo sequestrant for rocuronium and vecuronium.

Journal: Chemical communications (Cambridge, England)

Published: March 28, 2024

Hypoxia-mediated SUMOylation of FADD exacerbates endothelial cell injury via the RIPK1-RIPK3-MLKL signaling axis.

Hypoxia-mediated SUMOylation of FADD exacerbates endothelial cell injury via the RIPK1-RIPK3-MLKL signaling axis.

Journal: Cell death & disease

Published: February 10, 2024

Metabolite Neu5Ac triggers SLC3A2 degradation promoting vascular endothelial ferroptosis and aggravates atherosclerosis progression in ApoE-/-mice.

Metabolite Neu5Ac triggers SLC3A2 degradation promoting vascular endothelial ferroptosis and aggravates atherosclerosis progression in ApoE-/-mice.

Journal: Theranostics

Published: July 10, 2023

Sialic acids promote macrophage M1 polarization and atherosclerosis by upregulating ROS and autophagy blockage.

Sialic acids promote macrophage M1 polarization and atherosclerosis by upregulating ROS and autophagy blockage.

Journal: International immunopharmacology

Published: January 10, 2023

Tetramethylpyrazine Alleviates Endothelial Glycocalyx Degradation and Promotes Glycocalyx Restoration via TLR4/NF-κB/HPSE1 Signaling Pathway During Inflammation.

Tetramethylpyrazine Alleviates Endothelial Glycocalyx Degradation and Promotes Glycocalyx Restoration via TLR4/NF-κB/HPSE1 Signaling Pathway During Inflammation.

Journal: Frontiers in pharmacology

Published: October 09, 2021

Showing 1-11 of 11

Last Updated: 09/26/2026