Weidong Zhu
Department Of Otolaryngology Head And Neck Surgery
Shanghai, China
Shanghai, China
Last Updated: 09/26/2026
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Found 9 publications
Origins of biallelic inactivation of NF2 in neurofibromatosis type 2.
Journal: Neuro-oncology
Published: December 17, 2021
Age-dependent copy number variations of TP53 tumour suppressor gene associated with altered phosphorylation status of p53 protein in sporadic schwannomas.
Journal: Journal of neuro-oncology
Published: February 23, 2019
EYA1 mutations leads to Branchio-Oto syndrome in two Chinese Han deaf families.
Journal: International journal of pediatric otorhinolaryngology
Published: December 27, 2018
Synergistic effect of Nutlin-3 combined with MG-132 on schwannoma cells through restoration of merlin and p53 tumour suppressors.
Journal: EBioMedicine
Published: August 08, 2018
Intratympanic Gentamicin for Small Vestibular Schwannomas With Intractable Vertigo.
Journal: Otology & neurotology : official publication of the American Otological Society, American Neurotology Society [and] European Academy of Otology and Neurotology
Published: July 19, 2018
Impact of cochlear implantation on the management strategy of patients with neurofibromatosis type 2.
Journal: European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery
Published: March 22, 2018
Long-Term Hearing Preservation Outcomes for Small Vestibular Schwannomas: Retrosigmoid Removal Versus Observation.
Journal: Otology & neurotology : official publication of the American Otological Society, American Neurotology Society [and] European Academy of Otology and Neurotology
Published: January 10, 2018
Genetic and epigenetic patterns in patients with the head-and-neck paragangliomas associate with differential clinical characteristics.
Journal: Journal of cancer research and clinical oncology
Published: January 04, 2017
p53 performs an essential role in mediating the oncogenic stimulus triggered by loss of expression of neurofibromatosis type 2 during in vitro tumor progression.
Journal: Oncology letters
Published: September 15, 2015
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Last Updated: 09/26/2026