Weiwen Yan

The Key Laboratory Of Modern Toxicology Of Ministry Of Education, Jiangsu Key Laboratory Of Cancer Biomarkers
Nanjing, 211166, China

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 17 publications

The single-cell atlas of programmed cell death signature: A machine learning-based prognostic framework in breast cancer.

The single-cell atlas of programmed cell death signature: A machine learning-based prognostic framework in breast cancer.

Journal: Journal of biomedical research

Published: July 05, 2026

Bisphenol F disrupts lipophagy and lysosomal acidification via ATGL-SIRT1-PPARα signaling in NAFLD-like hepatic changes.

Bisphenol F disrupts lipophagy and lysosomal acidification via ATGL-SIRT1-PPARα signaling in NAFLD-like hepatic changes.

Journal: Ecotoxicology and environmental safety

Published: June 12, 2025

PTX3 alleviates hard metal-induced acute lung injury through potentiating efferocytosis.

PTX3 alleviates hard metal-induced acute lung injury through potentiating efferocytosis.

Journal: Ecotoxicology and environmental safety

Published: September 08, 2021

MiR-503 suppresses fibroblast activation and myofibroblast differentiation by targeting VEGFA and FGFR1 in silica-induced pulmonary fibrosis.

MiR-503 suppresses fibroblast activation and myofibroblast differentiation by targeting VEGFA and FGFR1 in silica-induced pulmonary fibrosis.

Journal: Journal of cellular and molecular medicine

Published: May 15, 2020

MiR-326 Inhibits Inflammation and Promotes Autophagy in Silica-Induced Pulmonary Fibrosis through Targeting TNFSF14 and PTBP1.

MiR-326 Inhibits Inflammation and Promotes Autophagy in Silica-Induced Pulmonary Fibrosis through Targeting TNFSF14 and PTBP1.

Journal: Chemical research in toxicology

Published: October 24, 2019

Aberrant expression of miR-125a-3p promotes fibroblast activation via Fyn/STAT3 pathway during silica-induced pulmonary fibrosis.

Aberrant expression of miR-125a-3p promotes fibroblast activation via Fyn/STAT3 pathway during silica-induced pulmonary fibrosis.

Journal: Toxicology

Published: October 28, 2018

The CDR1as/miR-7/TGFBR2 Axis Modulates EMT in Silica-Induced Pulmonary Fibrosis.

The CDR1as/miR-7/TGFBR2 Axis Modulates EMT in Silica-Induced Pulmonary Fibrosis.

Journal: Toxicological sciences : an official journal of the Society of Toxicology

Published: September 12, 2018

Quality of life and influencing factors of coal miners in Xuzhou, China.

Quality of life and influencing factors of coal miners in Xuzhou, China.

Journal: Journal of thoracic disease

Published: April 03, 2018

miR-1224-5p Mediates Mitochondrial Damage to Affect Silica-Induced Pulmonary Fibrosis by Targeting BECN1.

miR-1224-5p Mediates Mitochondrial Damage to Affect Silica-Induced Pulmonary Fibrosis by Targeting BECN1.

Journal: International journal of molecular sciences

Published: September 07, 2017

LRBA Gene Polymorphisms and Risk of Coal Workers' Pneumoconiosis: A Case-Control Study from China.

LRBA Gene Polymorphisms and Risk of Coal Workers' Pneumoconiosis: A Case-Control Study from China.

Journal: International journal of environmental research and public health

Published: August 03, 2017

Long non-coding RNA-ATB promotes EMT during silica-induced pulmonary fibrosis by competitively binding miR-200c.

Long non-coding RNA-ATB promotes EMT during silica-induced pulmonary fibrosis by competitively binding miR-200c.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: July 20, 2017

MiR-503 modulates epithelial-mesenchymal transition in silica-induced pulmonary fibrosis by targeting PI3K p85 and is sponged by lncRNA MALAT1.

MiR-503 modulates epithelial-mesenchymal transition in silica-induced pulmonary fibrosis by targeting PI3K p85 and is sponged by lncRNA MALAT1.

Journal: Scientific reports

Published: April 21, 2017

Showing 1-12 of 17

Last Updated: 09/26/2026