Weiyang Li

Beijing, 100069, China

Last Updated: 09/26/2026

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Found 14 publications

Neutrophil aconitate decarboxylase 1/itaconate axis suppresses extracellular traps via alkylating GSDMD and alleviates murine liver injury.

Neutrophil aconitate decarboxylase 1/itaconate axis suppresses extracellular traps via alkylating GSDMD and alleviates murine liver injury.

Journal: International immunopharmacology

Published: January 11, 2026

Pathogenic Ifit1+ neutrophils driven by IRF7 promote liver injury and represent a therapeutic target in acute liver failure.

Pathogenic Ifit1+ neutrophils driven by IRF7 promote liver injury and represent a therapeutic target in acute liver failure.

Journal: Hepatology (Baltimore, Md.)

Published: October 28, 2025

IQGAP1 participates in bone marrow-derived macrophage recruitment and involves in liver inflammation/fibrosis.

IQGAP1 participates in bone marrow-derived macrophage recruitment and involves in liver inflammation/fibrosis.

Journal: Journal of molecular medicine (Berlin, Germany)

Published: March 24, 2025

Neutrophil-secreted S100A8/A9 participates in fatty liver injury and fibrosis by promoting myofibroblast migration.

Neutrophil-secreted S100A8/A9 participates in fatty liver injury and fibrosis by promoting myofibroblast migration.

Journal: Journal of molecular medicine (Berlin, Germany)

Published: July 10, 2023

15-deoxy-Δ12,14-prostaglandin J2 relieved acute liver injury by inhibiting macrophage migration inhibitory factor expression via PPARγ in hepatocyte.

15-deoxy-Δ12,14-prostaglandin J2 relieved acute liver injury by inhibiting macrophage migration inhibitory factor expression via PPARγ in hepatocyte.

Journal: International immunopharmacology

Published: March 13, 2023

Single-cell RNA seq identifies Plg-RKT-PLG as signals inducing phenotypic transformation of scar-associated macrophage in liver fibrosis.

Single-cell RNA seq identifies Plg-RKT-PLG as signals inducing phenotypic transformation of scar-associated macrophage in liver fibrosis.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: November 07, 2022

Monocyte-derived Kupffer cells dominate in the Kupffer cell pool during liver injury.

Monocyte-derived Kupffer cells dominate in the Kupffer cell pool during liver injury.

Journal: Cell reports

Published: August 28, 2022

Necroptosis of macrophage is a key pathological feature in biliary atresia via GDCA/S1PR2/ZBP1/p-MLKL axis.

Necroptosis of macrophage is a key pathological feature in biliary atresia via GDCA/S1PR2/ZBP1/p-MLKL axis.

Journal: Cell death & disease

Published: July 30, 2022

Heterogeneity and Function of Kupffer Cells in Liver Injury.

Heterogeneity and Function of Kupffer Cells in Liver Injury.

Journal: Frontiers in immunology

Published: May 10, 2022

Cannabinoid Receptor 1 Participates in Liver Inflammation by Promoting M1 Macrophage Polarization via RhoA/NF-κB p65 and ERK1/2 Pathways, Respectively, in Mouse Liver Fibrogenesis.

Cannabinoid Receptor 1 Participates in Liver Inflammation by Promoting M1 Macrophage Polarization via RhoA/NF-κB p65 and ERK1/2 Pathways, Respectively, in Mouse Liver Fibrogenesis.

Journal: Frontiers in immunology

Published: June 30, 2017

miR-27b-3p, miR-181a-1-3p, and miR-326-5p are involved in the inhibition of macrophage activation in chronic liver injury.

miR-27b-3p, miR-181a-1-3p, and miR-326-5p are involved in the inhibition of macrophage activation in chronic liver injury.

Journal: Journal of molecular medicine (Berlin, Germany)

Published: February 17, 2017

Macrophage Migration Inhibitor Factor Upregulates MCP-1 Expression in an Autocrine Manner in Hepatocytes during Acute Mouse Liver Injury.

Macrophage Migration Inhibitor Factor Upregulates MCP-1 Expression in an Autocrine Manner in Hepatocytes during Acute Mouse Liver Injury.

Journal: Scientific reports

Published: February 15, 2016

Showing 1-12 of 14

Last Updated: 09/26/2026