Weiyuan Gong

Guangdong Provincial Key Laboratory Of Cell Microenvironment And Disease Research, Shenzhen Key Laboratory Of Cell Microenvironment
Shenzhen, China

Last Updated: 09/26/2026

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Found 12 publications

Sustained Hypoxia-Inducible Factor 1-Alpha Accumulation Disrupts the Articular Niche to Promote Osteoarthritis Pathogenesis.

Sustained Hypoxia-Inducible Factor 1-Alpha Accumulation Disrupts the Articular Niche to Promote Osteoarthritis Pathogenesis.

Journal: Aging cell
Published: April 03, 2026

Endothelin-1/endothelin B receptor signalling mediates Prx1+ skeletal stem cells senescence: A driver of osteoporotic bone loss.

Endothelin-1/endothelin B receptor signalling mediates Prx1+ skeletal stem cells senescence: A driver of osteoporotic bone loss.

Journal: Journal of orthopaedic translation
Published: August 28, 2025

The talin1-p53 axis inhibits osteocyte senescence to promote bone mass and mediate skeletal adaptation to mechanical stimulation.

The talin1-p53 axis inhibits osteocyte senescence to promote bone mass and mediate skeletal adaptation to mechanical stimulation.

Journal: Theranostics
Published: August 11, 2025

Spatially resolved single-cell-based histocytomorphometry for growth plate analysis across multiple species.

Spatially resolved single-cell-based histocytomorphometry for growth plate analysis across multiple species.

Journal: Journal of orthopaedic translation
Published: July 04, 2025

Global prevalence, incidence, and years lived with disability (YLDs) of osteoarthritis: trends from 1990 to 2021 and projections to 2050.

Global prevalence, incidence, and years lived with disability (YLDs) of osteoarthritis: trends from 1990 to 2021 and projections to 2050.

Journal: Journal of orthopaedic translation
Published: June 26, 2025

Inactivation of Tnf-α/Tnfr signaling attenuates progression of intervertebral disc degeneration in mice.

Inactivation of Tnf-α/Tnfr signaling attenuates progression of intervertebral disc degeneration in mice.

Journal: JOR spine
Published: June 17, 2024

Comparison of Kindlin-2 deficiency-stimulated osteoarthritis-like lesions induced by Prg4CreERT2 versus AggrecanCreERT2 transgene in mice.

Comparison of Kindlin-2 deficiency-stimulated osteoarthritis-like lesions induced by Prg4CreERT2 versus AggrecanCreERT2 transgene in mice.

Journal: Journal of orthopaedic translation
Published: December 29, 2022

Loss of Pinch Proteins Causes Severe Degenerative Disc Disease-Like Lesions in Mice.

Loss of Pinch Proteins Causes Severe Degenerative Disc Disease-Like Lesions in Mice.

Journal: Aging and disease
Published: November 27, 2022

Osteoarthritis: pathogenic signaling pathways and therapeutic targets.

Osteoarthritis: pathogenic signaling pathways and therapeutic targets.

Journal: Signal transduction and targeted therapy
Published: September 15, 2022

Pip5k1c Loss in Chondrocytes Causes Spontaneous Osteoarthritic Lesions in Aged Mice.

Pip5k1c Loss in Chondrocytes Causes Spontaneous Osteoarthritic Lesions in Aged Mice.

Journal: Aging and disease
Published: June 01, 2022

Kindlin-2 loss in condylar chondrocytes causes spontaneous osteoarthritic lesions in the temporomandibular joint in mice.

Kindlin-2 loss in condylar chondrocytes causes spontaneous osteoarthritic lesions in the temporomandibular joint in mice.

Journal: International journal of oral science
Published: January 10, 2022

Kindlin-2 deletion in osteoprogenitors causes severe chondrodysplasia and low-turnover osteopenia in mice.

Kindlin-2 deletion in osteoprogenitors causes severe chondrodysplasia and low-turnover osteopenia in mice.

Journal: Journal of orthopaedic translation
Published: June 28, 2021
Showing 1-12 of 12

Last Updated: 09/26/2026