Wenjian Li
Zhongshan East Road 361
Shijiazhuang, 050017, China
Shijiazhuang, 050017, China
Last Updated: 09/26/2026
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Found 13 publications
CD11c+CD69+ trained APCs in lungs mediate allergic asthma through increased H3K27ac.
Journal: International immunopharmacology
Published: April 24, 2025
Eliminating the invading extracellular and intracellular FnBp+ bacteria from respiratory epithelial cells by autophagy mediated through FnBp-Fn-Integrin α5β1 axis.
Journal: Frontiers in cellular and infection microbiology
Published: October 20, 2023
TRIM38 Induced in Respiratory Syncytial Virus-infected Cells Downregulates Type I Interferon Expression by Competing with TRIM25 to Bind RIG-I.
Journal: Inflammation
Published: September 02, 2023
Mycobacterium bovis BCG Given at Birth Followed by Inactivated Respiratory Syncytial Virus Vaccine Prevents Vaccine-Enhanced Disease by Promoting Trained Macrophages and Resident Memory T Cells.
Journal: Journal of virology
Published: February 13, 2023
Activation of the Chemokine Receptor CCR1 and Preferential Recruitment of Gαi Suppress RSV Replication: Implications for Developing Novel Respiratory Syncytial Virus Treatment Strategies.
Journal: Journal of virology
Published: November 01, 2022
Proline metabolism reprogramming of trained macrophages induced by early respiratory infection combined with allergen sensitization contributes to development of allergic asthma in childhood of mice.
Journal: Frontiers in immunology
Published: June 27, 2022
M Protein of Group a Streptococcus Plays an Essential Role in Inducing High Expression of A20 in Macrophages Resulting in the Downregulation of Inflammatory Response in Lung Tissue.
Journal: Frontiers in cellular and infection microbiology
Published: January 26, 2018
Respiratory Syncytial Virus Replication Is Promoted by Autophagy-Mediated Inhibition of Apoptosis.
Journal: Journal of virology
Published: December 21, 2017
CpG in Combination with an Inhibitor of Notch Signaling Suppresses Formalin-Inactivated Respiratory Syncytial Virus-Enhanced Airway Hyperresponsiveness and Inflammation by Inhibiting Th17 Memory Responses and Promoting Tissue-Resident Memory Cells in Lungs.
Journal: Journal of virology
Published: October 23, 2016
HCV core protein binds to gC1qR to induce A20 expression and inhibit cytokine production through MAPKs and NF-κB signaling pathways.
Journal: Oncotarget
Published: January 05, 2016
Group A Streptococcus induces less p65 nuclear translocation and non-classical nuclear factor kappa B activation in macrophages, which possibly leads to a weaker inflammatory response.
Journal: International journal of infectious diseases : IJID : official publication of the International Society for Infectious Diseases
Published: June 18, 2015
Last Updated: 09/26/2026