Wenjun Hao
Guangzhou, 510080, China
Last Updated: 09/26/2026
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Found 8 publications
IL-33-Induced TREM2+ Macrophages Promote Pathological New Bone Formation Through CREG1-IGF2R Axis in Ankylosing Spondylitis.
Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: January 15, 2025
Sex dimorphism of IL-17-secreting peripheral blood mononuclear cells in ankylosing spondylitis based on bioinformatics analysis and machine learning.
Journal: BMC musculoskeletal disorders
Published: March 19, 2024
Intermittent administration of PTH for the treatment of inflammatory bone loss does not enhance entheseal pathological new bone formation.
Journal: Biochemical and biophysical research communications
Published: January 18, 2024
Nucleolar protein PEXF controls ribosomal RNA synthesis and pluripotency exit.
Journal: Developmental cell
Published: January 16, 2024
Tenascin-C promotes endochondral ossification and fracture healing through Hedgehog and Hippo signaling.
Journal: Biochemical and biophysical research communications
Published: September 13, 2023
CXCL12/CXCR4-Rac1-mediated migration of osteogenic precursor cells contributes to pathological new bone formation in ankylosing spondylitis.
Journal: Science advances
Published: April 06, 2022
BRD4 promotes heterotopic ossification through upregulation of LncRNA MANCR.
Journal: Bone & joint research
Published: October 18, 2021
Tenascin-C-mediated suppression of extracellular matrix adhesion force promotes entheseal new bone formation through activation of Hippo signalling in ankylosing spondylitis.
Journal: Annals of the rheumatic diseases
Published: January 26, 2021
Showing 1-8 of 8
Last Updated: 09/26/2026