Wiebke Gerhardt-Kallenborn
Frankfurt Am Main, HE, Germany
Last Updated: 09/26/2026
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Found 24 publications
Redox-dependent activation of protein kinase G1α contributes to transient receptor potential cation channel subfamily V member 1-mediated acute nociceptive pain behavior.
Journal: Redox report : communications in free radical research
Published: August 25, 2025
Identification of a Chemical Probe for BLT2 Activation by Scaffold Hopping.
Journal: Journal of medicinal chemistry
Published: January 13, 2025
Inhibition of Phosphodiesterase 10A Alleviates Pain-like Behavior in Mice.
Journal: Anesthesiology
Published: November 06, 2024
Nox4-dependent upregulation of S100A4 after peripheral nerve injury modulates neuropathic pain processing.
Journal: Free radical biology & medicine
Published: January 19, 2021
Rab27a Contributes to the Processing of Inflammatory Pain in Mice.
Journal: Cells
Published: April 14, 2020
Neuropathic and cAMP-induced pain behavior is ameliorated in mice lacking CNGB1.
Journal: Neuropharmacology
Published: November 15, 2019
Redox regulation of soluble epoxide hydrolase does not affect pain behavior in mice.
Journal: Neuroscience letters
Published: November 13, 2019
Rab7-a novel redox target that modulates inflammatory pain processing.
Journal: Pain
Published: April 11, 2017
Last Updated: 09/26/2026