Xiaofei He

1 Xuefubei Street, Ouhai District
Wenzhou, China

Last Updated: 09/26/2026

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Found 13 publications

NSUN2-FOSB reciprocity facilitates leukemogenesis in an m5C-dependent manner by increasing BCL2L1 expression.

NSUN2-FOSB reciprocity facilitates leukemogenesis in an m5C-dependent manner by increasing BCL2L1 expression.

Journal: Haematologica

Published: September 25, 2025

RPL5 deficiency-induced ribosomal stress targets a select subset of proteins and inhibits the PI3K-Akt-mTOR signaling pathway to eradicate leukemia stem cells.

RPL5 deficiency-induced ribosomal stress targets a select subset of proteins and inhibits the PI3K-Akt-mTOR signaling pathway to eradicate leukemia stem cells.

Journal: Cell death & disease

Published: March 02, 2025

Loss of G-protein coupled receptor 68 in hematopoietic tissues enhances long-term hematopoietic stem cell function upon aging.

Loss of G-protein coupled receptor 68 in hematopoietic tissues enhances long-term hematopoietic stem cell function upon aging.

Journal: Stem cell research & therapy

Published: February 03, 2025

Genomic identification of a pair of multidrug-resistant but non-pathogenic Salmonella enterica serovar Goldcoast isolates in southeast China.

Genomic identification of a pair of multidrug-resistant but non-pathogenic Salmonella enterica serovar Goldcoast isolates in southeast China.

Journal: Frontiers in microbiology

Published: December 17, 2024

GPR68 supports AML cells through the calcium/calcineurin pro-survival pathway and confers chemoresistance by mediating glucose metabolic symbiosis.

GPR68 supports AML cells through the calcium/calcineurin pro-survival pathway and confers chemoresistance by mediating glucose metabolic symbiosis.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: May 28, 2024

Low expression of miR-182 caused by DNA hypermethylation accelerates acute lymphocyte leukemia development by targeting PBX3 and BCL2: miR-182 promoter methylation is a predictive marker for hypomethylation agents + BCL2 inhibitor venetoclax.

Low expression of miR-182 caused by DNA hypermethylation accelerates acute lymphocyte leukemia development by targeting PBX3 and BCL2: miR-182 promoter methylation is a predictive marker for hypomethylation agents + BCL2 inhibitor venetoclax.

Journal: Clinical epigenetics

Published: January 13, 2024

Low-dose hypomethylating agents cooperate with ferroptosis inducers to enhance ferroptosis by regulating the DNA methylation-mediated MAGEA6-AMPK-SLC7A11-GPX4 signaling pathway in acute myeloid leukemia.

Low-dose hypomethylating agents cooperate with ferroptosis inducers to enhance ferroptosis by regulating the DNA methylation-mediated MAGEA6-AMPK-SLC7A11-GPX4 signaling pathway in acute myeloid leukemia.

Journal: Experimental hematology & oncology

Published: October 14, 2023

The role of bile acid receptor TGR5 in regulating inflammatory signalling.

The role of bile acid receptor TGR5 in regulating inflammatory signalling.

Journal: Scandinavian journal of immunology

Published: August 09, 2023

Prognostic value analysis of cholesterol and cholesterol homeostasis related genes in breast cancer by Mendelian randomization and multi-omics machine learning.

Prognostic value analysis of cholesterol and cholesterol homeostasis related genes in breast cancer by Mendelian randomization and multi-omics machine learning.

Journal: Frontiers in oncology

Published: June 24, 2023

DNA hypermethylation-induced miR-182 silence targets BCL2 and HOXA9 to facilitate the self-renewal of leukemia stem cell, accelerate acute myeloid leukemia progression, and determine the sensitivity of BCL2 inhibitor venetoclax.

DNA hypermethylation-induced miR-182 silence targets BCL2 and HOXA9 to facilitate the self-renewal of leukemia stem cell, accelerate acute myeloid leukemia progression, and determine the sensitivity of BCL2 inhibitor venetoclax.

Journal: Theranostics

Published: July 23, 2022

Cyclosporine enhances the sensitivity to lenalidomide in MDS/AML in vitro.

Cyclosporine enhances the sensitivity to lenalidomide in MDS/AML in vitro.

Journal: Experimental hematology

Published: January 14, 2020

Whole body deletion of Gpr68 does not change hematopoietic stem cell function.

Whole body deletion of Gpr68 does not change hematopoietic stem cell function.

Journal: Stem cell research

Published: January 05, 2020

Showing 1-12 of 13

Last Updated: 09/26/2026