Xiaotian Fu

Key Laboratory Of Microbial Diversity Research And Application Of Hebei Province
Baoding, 071002, China

Last Updated: 09/26/2026

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Found 11 publications

Lipid droplets are potentially involved in bovine alphaherpesvirus 1 productive infection partially via stabilization of virion-associated proteins.

Lipid droplets are potentially involved in bovine alphaherpesvirus 1 productive infection partially via stabilization of virion-associated proteins.

Journal: Veterinary microbiology

Published: January 07, 2026

EEF1D signaling contributes to Bovine alpha herpesvirus 1 productive infection, potentially through regulation of viral replication compartments.

EEF1D signaling contributes to Bovine alpha herpesvirus 1 productive infection, potentially through regulation of viral replication compartments.

Journal: Veterinary microbiology

Published: April 23, 2025

The depletion of TFAM and p-β-catenin(S552) in mitochondria in response to BoAHV-1 productive infection leads to decreased mitochondrial biogenesis.

The depletion of TFAM and p-β-catenin(S552) in mitochondria in response to BoAHV-1 productive infection leads to decreased mitochondrial biogenesis.

Journal: Veterinary microbiology

Published: January 13, 2025

A B-cell epitope on the bovine herpesvirus 1 (BoAHV1) viral protein gC, referred to as PV116, is implicated in the generation of antibodies and the development of an ELISA kit for the detection of the virus antibody.

A B-cell epitope on the bovine herpesvirus 1 (BoAHV1) viral protein gC, referred to as PV116, is implicated in the generation of antibodies and the development of an ELISA kit for the detection of the virus antibody.

Journal: BMC microbiology

Published: November 13, 2024

HMGA1 Plays a Role in Counteracting DNA Damage Induced by BoHV-1 Productive Infection.

HMGA1 Plays a Role in Counteracting DNA Damage Induced by BoHV-1 Productive Infection.

Journal: International journal of molecular sciences

Published: November 04, 2024

53BP1, a known chromatin-associated factor that promotes DNA damage repair, is differentially modulated during bovine herpesvirus 1 infection in vitro and in vivo.

53BP1, a known chromatin-associated factor that promotes DNA damage repair, is differentially modulated during bovine herpesvirus 1 infection in vitro and in vivo.

Journal: Veterinary microbiology

Published: October 09, 2024

Bovine Herpesvirus 1 Productive Infection Led to Inactivation of Nrf2 Signaling through Diverse Approaches.

Bovine Herpesvirus 1 Productive Infection Led to Inactivation of Nrf2 Signaling through Diverse Approaches.

Journal: Oxidative medicine and cellular longevity

Published: March 31, 2019

The Differential Expression of Mitochondrial Function-Associated Proteins and Antioxidant Enzymes during Bovine Herpesvirus 1 Infection: A Potential Mechanism for Virus Infection-Induced Oxidative Mitochondrial Dysfunction.

The Differential Expression of Mitochondrial Function-Associated Proteins and Antioxidant Enzymes during Bovine Herpesvirus 1 Infection: A Potential Mechanism for Virus Infection-Induced Oxidative Mitochondrial Dysfunction.

Journal: Mediators of inflammation

Published: September 17, 2018

The Involvement of Histone H3 Acetylation in Bovine Herpesvirus 1 Replication in MDBK Cells.

The Involvement of Histone H3 Acetylation in Bovine Herpesvirus 1 Replication in MDBK Cells.

Journal: Viruses

Published: August 03, 2018

Induction of Oxidative DNA Damage in Bovine Herpesvirus 1 Infected Bovine Kidney Cells (MDBK Cells) and Human Tumor Cells (A549 Cells and U2OS Cells).

Induction of Oxidative DNA Damage in Bovine Herpesvirus 1 Infected Bovine Kidney Cells (MDBK Cells) and Human Tumor Cells (A549 Cells and U2OS Cells).

Journal: Viruses

Published: July 10, 2018

The synergistic antiviral effects of GSH in combination with acyclovir against BoHV-1 infection in vitro.

The synergistic antiviral effects of GSH in combination with acyclovir against BoHV-1 infection in vitro.

Journal: Acta virologica

Published: September 20, 2016

Showing 1-11 of 11

Last Updated: 09/26/2026