Ximeng Zhang

Guangdong Provincial Key Laboratory Of Infection Immunity & Inflammation
Shenzhen, 518000, China

Last Updated: 09/26/2026

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Found 9 publications

A CD40-OX40 co-stimulatory circuit orchestrates protective CD4+ T cell immunity in tuberculosis.

A CD40-OX40 co-stimulatory circuit orchestrates protective CD4+ T cell immunity in tuberculosis.

Journal: Molecular therapy : the journal of the American Society of Gene Therapy
Published: July 16, 2025

Mycobacterium tuberculosis IDH-PPARγ interaction suppresses GPX4 to drive macrophage ferroptosis and sustain persistent infection.

Mycobacterium tuberculosis IDH-PPARγ interaction suppresses GPX4 to drive macrophage ferroptosis and sustain persistent infection.

Journal: Nature communications
Published: July 16, 2025

SIRT7 remodels the cytoskeleton via RAC1 to enhance host resistance to Mycobacterium tuberculosis.

SIRT7 remodels the cytoskeleton via RAC1 to enhance host resistance to Mycobacterium tuberculosis.

Journal: mBio
Published: September 17, 2024

Granzyme A as biomarker for diagnosis in tuberculous pleural effusion.

Granzyme A as biomarker for diagnosis in tuberculous pleural effusion.

Journal: JCI insight
Published: July 29, 2024

Myriocin enhances the clearance of M. tuberculosis by macrophages through the activation of PLIN2.

Myriocin enhances the clearance of M. tuberculosis by macrophages through the activation of PLIN2.

Journal: mSphere
Published: June 26, 2024

Pathogenic role for CD101-negative neutrophils in the type I interferon-mediated immunopathogenesis of tuberculosis.

Pathogenic role for CD101-negative neutrophils in the type I interferon-mediated immunopathogenesis of tuberculosis.

Journal: Cell reports
Published: May 01, 2024

Mycobacterium tuberculosis-Induced Prostaglandin J2 and 15-Deoxy-Prostaglandin J2 Inhibit Inflammatory Signals in Human M1 Macrophages via a Negative Feedback Loop.

Mycobacterium tuberculosis-Induced Prostaglandin J2 and 15-Deoxy-Prostaglandin J2 Inhibit Inflammatory Signals in Human M1 Macrophages via a Negative Feedback Loop.

Journal: Journal of immunology (Baltimore, Md. : 1950)
Published: November 04, 2022

Mycobacterium tuberculosis Utilizes Host Histamine Receptor H1 to Modulate Reactive Oxygen Species Production and Phagosome Maturation via the p38MAPK-NOX2 Axis.

Mycobacterium tuberculosis Utilizes Host Histamine Receptor H1 to Modulate Reactive Oxygen Species Production and Phagosome Maturation via the p38MAPK-NOX2 Axis.

Journal: mBio
Published: August 24, 2022

Proteomics in Biomarker Discovery for Tuberculosis: Current Status and Future Perspectives.

Proteomics in Biomarker Discovery for Tuberculosis: Current Status and Future Perspectives.

Journal: Frontiers in microbiology
Published: December 29, 2021
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Last Updated: 09/26/2026