Xin Mou

208 East Huancheng Road
Hangzhou, 310003, China

Last Updated: 09/26/2026

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Found 20 publications

Retraction notice to "Chronic treatment with anti-GIPR mAb alone and combined with DPP-4 inhibitor correct obesity, dyslipidemia and nephropathy in rodent animals" [Life Sciences 269 (2021) 119038].

Retraction notice to "Chronic treatment with anti-GIPR mAb alone and combined with DPP-4 inhibitor correct obesity, dyslipidemia and nephropathy in rodent animals" [Life Sciences 269 (2021) 119038].

Journal: Life sciences

Published: March 11, 2026

Shenxiao decoction ameliorates podocyte injury in diabetic nephropathy via upregulating RUNX3 expression and inhibiting the JAK2/STAT3 signaling pathway.

Shenxiao decoction ameliorates podocyte injury in diabetic nephropathy via upregulating RUNX3 expression and inhibiting the JAK2/STAT3 signaling pathway.

Journal: Journal of ethnopharmacology

Published: November 19, 2025

Semaglutide and improved spinal fusion outcomes: a correspondence.

Semaglutide and improved spinal fusion outcomes: a correspondence.

Journal: International journal of surgery (London, England)

Published: August 27, 2025

Renshen Zhuye decoction ameliorates high-fat diet-induced obesity and insulin resistance by modulating gut microbiota and metabolic homeostasis.

Renshen Zhuye decoction ameliorates high-fat diet-induced obesity and insulin resistance by modulating gut microbiota and metabolic homeostasis.

Journal: Phytomedicine : international journal of phytotherapy and phytopharmacology

Published: January 20, 2025

Mmu-miR-664-5p contributes to high glucose-induced MPC5 podocyte injury via its target RUNX3.

Mmu-miR-664-5p contributes to high glucose-induced MPC5 podocyte injury via its target RUNX3.

Journal: Toxicology research

Published: January 09, 2025

Chronic treatment with anti-GIPR mAb alone and combined with DPP-4 inhibitor correct obesity, dyslipidemia and nephropathy in rodent animals.

Chronic treatment with anti-GIPR mAb alone and combined with DPP-4 inhibitor correct obesity, dyslipidemia and nephropathy in rodent animals.

Journal: Life sciences

Published: November 09, 2020

Amygdalin alleviates renal injury by suppressing inflammation, oxidative stress and fibrosis in streptozotocin-induced diabetic rats.

Amygdalin alleviates renal injury by suppressing inflammation, oxidative stress and fibrosis in streptozotocin-induced diabetic rats.

Journal: Life sciences

Published: October 27, 2020

Acarbose ameliorates spontaneous type‑2 diabetes in db/db mice by inhibiting PDX‑1 methylation.

Acarbose ameliorates spontaneous type‑2 diabetes in db/db mice by inhibiting PDX‑1 methylation.

Journal: Molecular medicine reports

Published: June 17, 2020

Hepatic Proteomic Changes and Sirt1/AMPK Signaling Activation by Oxymatrine Treatment in Rats With Non-alcoholic Steatosis.

Hepatic Proteomic Changes and Sirt1/AMPK Signaling Activation by Oxymatrine Treatment in Rats With Non-alcoholic Steatosis.

Journal: Frontiers in pharmacology

Published: November 05, 2019

A bioinformatics and network pharmacology approach to the mechanisms of action of Shenxiao decoction for the treatment of diabetic nephropathy.

A bioinformatics and network pharmacology approach to the mechanisms of action of Shenxiao decoction for the treatment of diabetic nephropathy.

Journal: Phytomedicine : international journal of phytotherapy and phytopharmacology

Published: September 13, 2019

In silico prediction and validation of potential therapeutic genes in pancreatic β-cells associated with type 2 diabetes.

In silico prediction and validation of potential therapeutic genes in pancreatic β-cells associated with type 2 diabetes.

Journal: Experimental and therapeutic medicine

Published: August 06, 2019

A novel identified circular RNA, circ_0000491, aggravates the extracellular matrix of diabetic nephropathy glomerular mesangial cells through suppressing miR‑101b by targeting TGFβRI.

A novel identified circular RNA, circ_0000491, aggravates the extracellular matrix of diabetic nephropathy glomerular mesangial cells through suppressing miR‑101b by targeting TGFβRI.

Journal: Molecular medicine reports

Published: August 06, 2019

Showing 1-12 of 20

Last Updated: 09/26/2026