Xintong Dai

State Key Laboratory Of Medicinal Chemical Biology, College Of Pharmacy And Tianjin Key Laboratory Of Molecular Drug Research
Changhongjie, 300350, China

Last Updated: 09/26/2026

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Found 19 publications

HPD is a copper-binding protein that interacts with DLAT to promote colorectal cancer cuproptosis under copper stress.

HPD is a copper-binding protein that interacts with DLAT to promote colorectal cancer cuproptosis under copper stress.

Journal: Cell reports

Published: January 19, 2026

4-Hydroxychalcone Exert the Anti-cancer Potential in Colorectal Cancer Through Targeting CDK6.

4-Hydroxychalcone Exert the Anti-cancer Potential in Colorectal Cancer Through Targeting CDK6.

Journal: Biological procedures online

Published: January 13, 2026

Single-cell RNA sequencing and proteomics uncover SIRPα-CD47 immune checkpoint and glycolysis-driven immune evasion in cardiac myxoma.

Single-cell RNA sequencing and proteomics uncover SIRPα-CD47 immune checkpoint and glycolysis-driven immune evasion in cardiac myxoma.

Journal: Frontiers in immunology

Published: June 24, 2025

USP30 senses serine/glycine levels to regulate serine biosynthesis and colorectal tumorigenesis by deubiquitinating FTO.

USP30 senses serine/glycine levels to regulate serine biosynthesis and colorectal tumorigenesis by deubiquitinating FTO.

Journal: Cell death and differentiation

Published: June 09, 2025

HPD is an m6A Methyltransferase that Protects Colorectal Cancer Cells from Ferroptotic Cell Death by m6A Methylating SLC7A11/GPX4.

HPD is an m6A Methyltransferase that Protects Colorectal Cancer Cells from Ferroptotic Cell Death by m6A Methylating SLC7A11/GPX4.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: May 12, 2025

HPD is an RNA-Binding Protein Sustaining Ovarian Cancer Cell Glycolysis, Tumor Growth, and Drug Resistance.

HPD is an RNA-Binding Protein Sustaining Ovarian Cancer Cell Glycolysis, Tumor Growth, and Drug Resistance.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: March 03, 2025

The non-metabolic function of 6PGD coordinates CCNA2 and HMGA2 expression to drive colorectal cancer progression and drug response.

The non-metabolic function of 6PGD coordinates CCNA2 and HMGA2 expression to drive colorectal cancer progression and drug response.

Journal: Journal of experimental & clinical cancer research : CR

Published: January 15, 2025

Correction: Targeting FTO induces colorectal cancer ferroptotic cell death by decreasing SLC7A11/ GPX4 expression.

Correction: Targeting FTO induces colorectal cancer ferroptotic cell death by decreasing SLC7A11/ GPX4 expression.

Journal: Journal of experimental & clinical cancer research : CR

Published: May 01, 2024

SF3B3-regulated mTOR alternative splicing promotes colorectal cancer progression and metastasis.

SF3B3-regulated mTOR alternative splicing promotes colorectal cancer progression and metastasis.

Journal: Journal of experimental & clinical cancer research : CR

Published: March 05, 2024

Targeting FTO induces colorectal cancer ferroptotic cell death by decreasing SLC7A11/GPX4 expression.

Targeting FTO induces colorectal cancer ferroptotic cell death by decreasing SLC7A11/GPX4 expression.

Journal: Journal of experimental & clinical cancer research : CR

Published: January 10, 2024

A model for identification of potential phase-separated proteins based on protein sequence, structure and cellular distribution.

A model for identification of potential phase-separated proteins based on protein sequence, structure and cellular distribution.

Journal: International journal of biological macromolecules

Published: March 28, 2023

Microbiota-derived imidazole propionate inhibits type 2 diabetic skin wound healing by targeting SPNS2-mediated S1P transport.

Microbiota-derived imidazole propionate inhibits type 2 diabetic skin wound healing by targeting SPNS2-mediated S1P transport.

Journal: iScience

Published: February 07, 2023

Showing 1-12 of 19

Last Updated: 09/26/2026