Xuqing Shen

Shanghai, China

Last Updated: 09/26/2026

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Found 16 publications

Body temperature drives azole tolerance in Candida albicans by hindering the autophagic degradation of Erg11.

Body temperature drives azole tolerance in Candida albicans by hindering the autophagic degradation of Erg11.

Journal: Microbiological research

Published: December 20, 2025

Galactose Inhibits the Translation of Erg1 that Enhances the Antifungal Activities of Azoles Against Candida albicans.

Galactose Inhibits the Translation of Erg1 that Enhances the Antifungal Activities of Azoles Against Candida albicans.

Journal: Antibiotics (Basel, Switzerland)

Published: June 07, 2025

Extracellular Hsp90 of Candida albicans contributes to the virulence of the pathogen by activating the NF-κB signaling pathway and inducing macrophage pyroptosis.

Extracellular Hsp90 of Candida albicans contributes to the virulence of the pathogen by activating the NF-κB signaling pathway and inducing macrophage pyroptosis.

Journal: Microbiological research

Published: September 10, 2024

Targeting pancreatic cancer glutamine dependency confers vulnerability to GPX4-dependent ferroptosis.

Targeting pancreatic cancer glutamine dependency confers vulnerability to GPX4-dependent ferroptosis.

Journal: Cell reports. Medicine

Published: January 30, 2024

Tumor cell-intrinsic epigenetic dysregulation shapes cancer-associated fibroblasts heterogeneity to metabolically support pancreatic cancer.

Tumor cell-intrinsic epigenetic dysregulation shapes cancer-associated fibroblasts heterogeneity to metabolically support pancreatic cancer.

Journal: Cancer cell

Published: August 15, 2023

Clostridium butyricum and its metabolite butyrate promote ferroptosis susceptibility in pancreatic ductal adenocarcinoma.

Clostridium butyricum and its metabolite butyrate promote ferroptosis susceptibility in pancreatic ductal adenocarcinoma.

Journal: Cellular oncology (Dordrecht, Netherlands)

Published: May 15, 2023

Spatiotemporal role of SETD2-H3K36me3 in murine pancreatic organogenesis.

Spatiotemporal role of SETD2-H3K36me3 in murine pancreatic organogenesis.

Journal: Cell reports

Published: January 04, 2023

The diverse pancreatic tumor cell-intrinsic response to IFNγ is determined by epigenetic heterogeneity.

The diverse pancreatic tumor cell-intrinsic response to IFNγ is determined by epigenetic heterogeneity.

Journal: Cancer letters

Published: January 02, 2023

Oncogenic KRAS triggers metabolic reprogramming in pancreatic ductal adenocarcinoma.

Oncogenic KRAS triggers metabolic reprogramming in pancreatic ductal adenocarcinoma.

Journal: Journal of translational internal medicine

Published: November 15, 2022

Tumor Cell-Intrinsic SETD2 Deficiency Reprograms Neutrophils to Foster Immune Escape in Pancreatic Tumorigenesis.

Tumor Cell-Intrinsic SETD2 Deficiency Reprograms Neutrophils to Foster Immune Escape in Pancreatic Tumorigenesis.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: May 18, 2022

Glycolysis addiction compensating for a defective pentose phosphate pathway confers gemcitabine sensitivity in SETD2-deficient pancreatic cancer.

Glycolysis addiction compensating for a defective pentose phosphate pathway confers gemcitabine sensitivity in SETD2-deficient pancreatic cancer.

Journal: Biochemical and biophysical research communications

Published: April 30, 2022

Ketogenesis acts as an endogenous protective programme to restrain inflammatory macrophage activation during acute pancreatitis.

Ketogenesis acts as an endogenous protective programme to restrain inflammatory macrophage activation during acute pancreatitis.

Journal: EBioMedicine

Published: November 18, 2021

Showing 1-12 of 16

Last Updated: 09/26/2026