Yafan Liu

State Key Laboratory Of Experimental Hematology
Tianjin, China

Last Updated: 09/26/2026

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Found 14 publications

Gas-6 Scavenges Anionic Phospholipid-Expressing Microparticles and Mitigates TBI-Induced Endotheliopathy and Coagulopathy in Mice.

Gas-6 Scavenges Anionic Phospholipid-Expressing Microparticles and Mitigates TBI-Induced Endotheliopathy and Coagulopathy in Mice.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: August 06, 2026

Extracellular Mitochondria Mediate Endothelial Injury After Traumatic Brain Injury.

Extracellular Mitochondria Mediate Endothelial Injury After Traumatic Brain Injury.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: June 25, 2026

Developmental Endothelial Locus 1 Reduces Traumatic Brain Injury-Induced Endotheliopathy and Dysregulated Coagulation in Mice.

Developmental Endothelial Locus 1 Reduces Traumatic Brain Injury-Induced Endotheliopathy and Dysregulated Coagulation in Mice.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: July 24, 2025

Bispecific self-assembled peptides as supra-growth factors for preventing endotheliopathy and improving survival of traumatic brain injury in mice.

Bispecific self-assembled peptides as supra-growth factors for preventing endotheliopathy and improving survival of traumatic brain injury in mice.

Journal: Journal of nanobiotechnology

Published: May 24, 2025

Bone marrow-derived emergency monopoiesis drives brain-lung axis injury after traumatic brain injury via IL-1.

Bone marrow-derived emergency monopoiesis drives brain-lung axis injury after traumatic brain injury via IL-1.

Journal: Journal of neuroinflammation

Published: May 24, 2025

Neutrophil extracellular traps induce endothelial damage and exacerbate vasospasm in traumatic brain injury.

Neutrophil extracellular traps induce endothelial damage and exacerbate vasospasm in traumatic brain injury.

Journal: Theranostics

Published: April 14, 2025

Corrigendum to "Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury" [International Immunopharmacology 130 (2024) 111674].

Corrigendum to "Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury" [International Immunopharmacology 130 (2024) 111674].

Journal: International immunopharmacology

Published: March 27, 2025

β2 integrin regulates neutrophil trans endothelial migration following traumatic brain injury.

β2 integrin regulates neutrophil trans endothelial migration following traumatic brain injury.

Journal: Cell communication and signaling : CCS

Published: November 08, 2024

Extracellular mitochondria contribute to acute lung injury via disrupting macrophages after traumatic brain injury.

Extracellular mitochondria contribute to acute lung injury via disrupting macrophages after traumatic brain injury.

Journal: Journal of neuroinflammation

Published: October 20, 2024

Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury.

Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury.

Journal: International immunopharmacology

Published: November 27, 2023

Brain-derived extracellular vesicles mediate traumatic brain injury associated multi-organ damage.

Brain-derived extracellular vesicles mediate traumatic brain injury associated multi-organ damage.

Journal: Biochemical and biophysical research communications

Published: April 02, 2023

Transcription factors in megakaryocytes and platelets.

Transcription factors in megakaryocytes and platelets.

Journal: Frontiers in immunology

Published: January 09, 2023

Showing 1-12 of 14

Last Updated: 09/26/2026