Yanhong Xing

Jiangsu Province Key Laboratory Of Anesthesiology, Jiangsu Province Key Laboratory Of Anesthesia And Analgesia Application Technology, NMPA Key Laboratory For Research And Evaluation Of Narcotic And Psychotropic Drugs
Xuzhou, 221004, China

Last Updated: 09/26/2026

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Found 14 publications

Development of Knockout Cardiac Muscle Cell Lines Using Integrase-Deficient Lentivirus-Mediated CRISPR/Cas9 Gene Editing.

Development of Knockout Cardiac Muscle Cell Lines Using Integrase-Deficient Lentivirus-Mediated CRISPR/Cas9 Gene Editing.

Journal: Biochemical genetics

Published: February 27, 2025

MCOLN1/TRPML1 in the lysosome: a promising target for autophagy modulation in diverse diseases.

MCOLN1/TRPML1 in the lysosome: a promising target for autophagy modulation in diverse diseases.

Journal: Autophagy

Published: March 24, 2024

Lysosomes finely control macrophage inflammatory function via regulating the release of lysosomal Fe2+ through TRPML1 channel.

Lysosomes finely control macrophage inflammatory function via regulating the release of lysosomal Fe2+ through TRPML1 channel.

Journal: Nature communications

Published: January 10, 2024

Tumor necrosis factor α-induced protein 8-like-2 controls microglia phenotype via metabolic reprogramming in BV2 microglial cells and responses to neuropathic pain.

Tumor necrosis factor α-induced protein 8-like-2 controls microglia phenotype via metabolic reprogramming in BV2 microglial cells and responses to neuropathic pain.

Journal: The international journal of biochemistry & cell biology

Published: June 16, 2023

Microglia Sing the Prelude of Neuroinflammation-Associated Depression.

Microglia Sing the Prelude of Neuroinflammation-Associated Depression.

Journal: Molecular neurobiology

Published: June 14, 2023

Tumor necrosis factor-α-induced protein 8-like 2 alleviates morphine antinociceptive tolerance through reduction of ROS-mediated apoptosis and MAPK/NF-κB signaling pathways.

Tumor necrosis factor-α-induced protein 8-like 2 alleviates morphine antinociceptive tolerance through reduction of ROS-mediated apoptosis and MAPK/NF-κB signaling pathways.

Journal: Neuropharmacology

Published: December 11, 2022

Targeting MCOLN1/TRPML1 channels to protect against ischemia-reperfusion injury by restoring the inhibited autophagic flux in cardiomyocytes.

Targeting MCOLN1/TRPML1 channels to protect against ischemia-reperfusion injury by restoring the inhibited autophagic flux in cardiomyocytes.

Journal: Autophagy

Published: May 02, 2022

TRPML1-induced autophagy inhibition triggers mitochondrial mediated apoptosis.

TRPML1-induced autophagy inhibition triggers mitochondrial mediated apoptosis.

Journal: Cancer letters

Published: March 13, 2022

Autophagy inhibition mediated by MCOLN1/TRPML1 suppresses cancer metastasis via regulating a ROS-driven TP53/p53 pathway.

Autophagy inhibition mediated by MCOLN1/TRPML1 suppresses cancer metastasis via regulating a ROS-driven TP53/p53 pathway.

Journal: Autophagy

Published: December 08, 2021

Stimulating TRPM7 suppresses cancer cell proliferation and metastasis by inhibiting autophagy.

Stimulating TRPM7 suppresses cancer cell proliferation and metastasis by inhibiting autophagy.

Journal: Cancer letters

Published: September 15, 2021

Blunting TRPML1 channels protects myocardial ischemia/reperfusion injury by restoring impaired cardiomyocyte autophagy.

Blunting TRPML1 channels protects myocardial ischemia/reperfusion injury by restoring impaired cardiomyocyte autophagy.

Journal: Basic research in cardiology

Published: September 14, 2021

MCOLN1/TRPML1 finely controls oncogenic autophagy in cancer by mediating zinc influx.

MCOLN1/TRPML1 finely controls oncogenic autophagy in cancer by mediating zinc influx.

Journal: Autophagy

Published: April 23, 2021

Showing 1-12 of 14

Last Updated: 09/26/2026