Yash T. Katakia
Pilani, RJ, India
Last Updated: 09/26/2026
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Found 18 publications
Splicing factor proline- and glutamine-rich (SFPQ) protein causes transcriptional repression of SNAIL to counteract TGF-β signaling.
Journal: Biochimica et biophysica acta. Gene regulatory mechanisms
Published: December 03, 2025
Pharmacological Blockade of Hypoxia-Driven Autophagy: A Therapeutic Strategy to Limit Endothelial Activation and Thrombosis.
Journal: ACS pharmacology & translational science
Published: November 25, 2025
Histone H4K5 deacetylation by enhanced HDAC1 expression drives endothelial inflammation in diabetes through repression of the KLF2-eNOS axis.
Journal: Human cell
Published: November 22, 2025
Vitamin D attenuates PCSK9-driven phenotypic switch of vascular smooth muscle cells and neointimal hyperplasia via SIRT6 activation.
Journal: Biochimica et biophysica acta. Molecular basis of disease
Published: July 25, 2025
Regulation of shelterin proteins TERF2IP and TRF2 by the MLL2-H3K4me3-p65 axis drives hyperglycemia-dependent endothelial senescence.
Journal: International journal of biological macromolecules
Published: December 02, 2024
Inhibition of autophagy in platelets as a therapeutic strategy preventing hypoxia induced thrombosis.
Journal: Scientific reports
Published: September 17, 2024
New epigenome players in the regulation of PCSK9-H3K4me3 and H3K9ac alterations by statin in hypercholesterolemia.
Journal: Journal of lipid research
Published: July 01, 2024
S-nitrosylation of EZH2 alters PRC2 assembly, methyltransferase activity, and EZH2 stability to maintain endothelial homeostasis.
Journal: Nature communications
Published: February 08, 2024
Angular difference in human coronary artery governs endothelial cell structure and function.
Journal: Communications biology
Published: February 26, 2022
Elevated H3K4me3 Through MLL2-WDR82 upon Hyperglycemia Causes Jagged Ligand Dependent Notch Activation to Interplay with Differentiation State of Endothelial Cells.
Journal: Frontiers in cell and developmental biology
Published: December 19, 2021
Intermittent High Glucose Elevates Nuclear Localization of EZH2 to Cause H3K27me3-Dependent Repression of KLF2 Leading to Endothelial Inflammation.
Journal: Cells
Published: August 10, 2021
Last Updated: 09/26/2026